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1
Two-pore K+ channels, NO and metabolic inhibition.双孔钾通道、一氧化氮与代谢抑制
Biochem Biophys Res Commun. 2007 Nov 9;363(1):194-6. doi: 10.1016/j.bbrc.2007.08.131. Epub 2007 Aug 31.
2
AMP-activated protein kinase mediates preconditioning in cardiomyocytes by regulating activity and trafficking of sarcolemmal ATP-sensitive K(+) channels.AMP 激活的蛋白激酶通过调节肌膜 ATP 敏感性钾通道的活性和转运来介导心肌细胞的预处理。
J Cell Physiol. 2007 Jan;210(1):224-36. doi: 10.1002/jcp.20862.
3
Both metabolic inhibition and mitochondrial K(ATP) channel opening are myoprotective and initiate a compensatory sarcolemmal outward membrane current.代谢抑制和线粒体ATP敏感性钾通道开放均具有心肌保护作用，并引发补偿性肌膜外向膜电流。
Circulation. 2003 Sep 9;108 Suppl 1:II341-7. doi: 10.1161/01.cir.0000087431.91602.e2.
4
Hypoxia-induced preconditioning in adult stimulated cardiomyocytes is mediated by the opening and trafficking of sarcolemmal KATP channels.缺氧诱导的成年受刺激心肌细胞预处理是由肌膜ATP敏感性钾通道的开放和转运介导的。
FASEB J. 2004 Jun;18(9):1046-8. doi: 10.1096/fj.04-1602fje. Epub 2004 Apr 14.
5
Differential sensitivity of atrial and ventricular K(ATP) channels to metabolic inhibition.心房和心室K（ATP）通道对代谢抑制的差异敏感性。
Cardiovasc Res. 2003 Feb;57(2):468-76. doi: 10.1016/s0008-6363(02)00715-0.
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(-)-(9S)-9-(3-Bromo-4-fluorophenyl)-2,3,5,6,7,9-hexahydrothieno[3,2-b]quinolin-8(4H)-one 1,1-dioxide (A-278637): a novel ATP-sensitive potassium channel opener efficacious in suppressing urinary bladder contractions. I. In vitro characterization.(-)-(9S)-9-(3-溴-4-氟苯基)-2,3,5,6,7,9-六氢噻吩并[3,2-b]喹啉-8(4H)-酮 1,1-二氧化物（A-278637）：一种新型的ATP敏感性钾通道开放剂，对抑制膀胱收缩有效。I. 体外特性研究
J Pharmacol Exp Ther. 2002 Oct;303(1):379-86. doi: 10.1124/jpet.102.034538.
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Effect of anoxic preconditioning on ATP-sensitive potassium channels in guinea-pig ventricular myocytes.缺氧预处理对豚鼠心室肌细胞中ATP敏感性钾通道的影响。
Pflugers Arch. 2000 Apr;439(6):808-13. doi: 10.1007/s004249900200.
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High glucose protects single beating adult cardiomyocytes against hypoxia.高糖可保护成年单搏动心肌细胞免受缺氧损伤。
Biochem Biophys Res Commun. 2006 Mar 3;341(1):57-66. doi: 10.1016/j.bbrc.2005.12.147. Epub 2006 Jan 6.
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Effect of ajmaline on transient outward current in rat ventricular myocytes.阿义马林对大鼠心室肌细胞瞬时外向电流的影响。
Gen Physiol Biophys. 2005 Mar;24(1):27-45.
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Intracellular signalling mechanism responsible for modulation of sarcolemmal ATP-sensitive potassium channels by nitric oxide in ventricular cardiomyocytes.一氧化氮调控心室心肌细胞肌膜ATP敏感性钾通道的细胞内信号传导机制。
J Physiol. 2014 Mar 1;592(5):971-90. doi: 10.1113/jphysiol.2013.264697. Epub 2013 Nov 25.
引用本文的文献
1
Nitric Oxide and Mechano-Electrical Transduction in Cardiomyocytes.一氧化氮与心肌细胞的机械电转导
Front Physiol. 2020 Dec 15;11:606740. doi: 10.3389/fphys.2020.606740. eCollection 2020.
2
Regulation of two-pore-domain (K2P) potassium leak channels by the tyrosine kinase inhibitor genistein.酪氨酸激酶抑制剂金雀异黄素对双孔结构域（K2P）钾离子渗漏通道的调控作用
Br J Pharmacol. 2008 Aug;154(8):1680-90. doi: 10.1038/bjp.2008.213. Epub 2008 Jun 2.
本文引用的文献
1
Physiology and pharmacology of two-pore domain potassium channels.双孔结构域钾通道的生理学与药理学
Curr Pharm Des. 2005;11(21):2717-36. doi: 10.2174/1381612054546824.
2
Pancreatic two P domain K+ channels TALK-1 and TALK-2 are activated by nitric oxide and reactive oxygen species.胰腺双P结构域钾通道TALK-1和TALK-2可被一氧化氮和活性氧激活。
J Physiol. 2005 Jan 1;562(Pt 1):235-44. doi: 10.1113/jphysiol.2004.071266. Epub 2004 Oct 28.
3
The 2P-domain K+ channels: role in apoptosis and tumorigenesis.2P结构域钾离子通道：在细胞凋亡和肿瘤发生中的作用。
Pflugers Arch. 2004 Jun;448(3):261-73. doi: 10.1007/s00424-004-1255-8. Epub 2004 May 5.
4
Both metabolic inhibition and mitochondrial K(ATP) channel opening are myoprotective and initiate a compensatory sarcolemmal outward membrane current.代谢抑制和线粒体ATP敏感性钾通道开放均具有心肌保护作用，并引发补偿性肌膜外向膜电流。
Circulation. 2003 Sep 9;108 Suppl 1:II341-7. doi: 10.1161/01.cir.0000087431.91602.e2.
5
Apoptosis recruits two-pore domain potassium channels used for homeostatic volume regulation.
Am J Physiol Cell Physiol. 2002 Mar;282(3):C588-94. doi: 10.1152/ajpcell.00365.2001.
6
Molecular and functional properties of two-pore-domain potassium channels.双孔结构域钾通道的分子与功能特性
Am J Physiol Renal Physiol. 2000 Nov;279(5):F793-801. doi: 10.1152/ajprenal.2000.279.5.F793.
7
Priming effect of adenosine on K(ATP) currents in intact ventricular myocytes: implications for preconditioning.
Am J Physiol. 1997 Oct;273(4):H1637-43. doi: 10.1152/ajpheart.1997.273.4.H1637.
8
Cloning, functional expression and brain localization of a novel unconventional outward rectifier K+ channel.一种新型非传统外向整流钾通道的克隆、功能表达及脑定位
EMBO J. 1996 Dec 16;15(24):6854-62.
9
Electrophysiological and functional effects of adenosine on ventricular myocytes of various mammalian species.腺苷对不同哺乳动物物种心室肌细胞的电生理和功能影响。
Am J Physiol. 1996 Oct;271(4 Pt 1):C1233-43. doi: 10.1152/ajpcell.1996.271.4.C1233.
10
Synergistic modulation of ATP-sensitive K+ currents by protein kinase C and adenosine. Implications for ischemic preconditioning.蛋白激酶C和腺苷对ATP敏感性钾电流的协同调节作用。对缺血预处理的意义。
Circ Res. 1996 Mar;78(3):443-54. doi: 10.1161/01.res.78.3.443.
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