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淀粉样前体蛋白片段C99的蛋白质组学分析:在酵母中的表达

Proteomic analysis of the amyloid precursor protein fragment C99: expression in yeast.

作者信息

Sparvero Louis J, Patz Sarah, Brodsky Jeffrey L, Coughlan Christina M

机构信息

Department of Surgery, Center for Bioengineering, Mass Spectrometry Facility, University of Pittsburgh, Pittsburgh, PA 15261, USA.

出版信息

Anal Biochem. 2007 Nov 15;370(2):162-70. doi: 10.1016/j.ab.2007.07.033. Epub 2007 Aug 10.

Abstract

The accumulation and aggregation of fragments of amyloid precursor protein (APP) are central to the development of Alzheimer's disease. The production of the small fragment C99 is thought to form the rate-limiting step in the APP processing pathway, which can lead to the production of the toxic Abeta peptide. It has also been suggested that the proteasome contributes to APP catabolism. While the identities and aggregation propensities of many APP fragments have been studied in vitro, the sequences, structures, and cellular sources of fragments generated in vivo remains poorly elucidated. To better identify the specific APP fragments generated in vivo and to elucidate the role of the proteasome in APP processing, we developed a C99 yeast expression system. Using Zip Tip immunocapture, a specific anti-Abeta antiserum (6E10), and matrix-assisted laser desorption ionization- time of flight mass spectrometry, we identified over one dozen APP-generated peptide fragments in wild-type yeast (PRE1PRE2) and over three dozen unique fragments in proteasome mutant cells (pre1- 1pre2-1) expressing C99. Based on the identities of the immunocaptured species, we propose that defects in proteasome function are compensated by other proteases and that the combination of techniques described here will be invaluable to further delineate the APP processing pathway in vivo.

摘要

淀粉样前体蛋白(APP)片段的积累和聚集是阿尔茨海默病发展的核心。小片段C99的产生被认为是APP加工途径中的限速步骤,这可能导致有毒的β淀粉样肽的产生。也有人提出蛋白酶体有助于APP的分解代谢。虽然许多APP片段的身份和聚集倾向已在体外进行了研究,但体内产生的片段的序列、结构和细胞来源仍不清楚。为了更好地识别体内产生的特定APP片段并阐明蛋白酶体在APP加工中的作用,我们开发了一种C99酵母表达系统。使用Zip Tip免疫捕获、特异性抗β淀粉样抗血清(6E10)和基质辅助激光解吸电离飞行时间质谱,我们在野生型酵母(PRE1PRE2)中鉴定出十多种APP产生的肽片段,在表达C99的蛋白酶体突变细胞(pre1-1pre2-1)中鉴定出三十多种独特片段。基于免疫捕获物种的身份,我们提出蛋白酶体功能缺陷由其他蛋白酶补偿,并且这里描述的技术组合对于进一步阐明体内APP加工途径将是非常有价值的。

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