Gefen Amit
Department of Biomedical Engineering, Faculty of Engineering, Tel Aviv University, Tel Aviv, Israel.
Int Wound J. 2007 Sep;4(3):222-31. doi: 10.1111/j.1742-481X.2007.00330.x.
Sitting-acquired pressure ulcers (SAPU) in permanent wheelchair users with traumatic or non traumatic disorders of the central nervous system (CNS) are a great medical challenge. The purpose of this review is to summarise what is currently known concerning the aetiology of SAPU, particularly in its severe form, which may now be classified as a 'deep tissue injury' according to the US National Pressure Ulcer Advisory Panel. Specifically, this review focuses on biomechanical aspects of deep SAPU and describes the relevant bioengineering methodologies and research evidence. It discusses the unique biomechanical conditions in deep tissues, which are caused by chronic sitting associated with CNS disorders, and overall, the present review indicates that avoiding interface pressures above 32 mmHg in such patients is not necessarily a 'pressure relief.'
患有中枢神经系统(CNS)创伤性或非创伤性疾病的永久性轮椅使用者所发生的坐姿性压疮(SAPU)是一项重大的医学挑战。本综述的目的是总结目前已知的关于SAPU病因的知识,尤其是其严重形式,根据美国国家压疮咨询小组的分类,这种严重形式现在可能被归类为“深部组织损伤”。具体而言,本综述聚焦于深部SAPU的生物力学方面,并描述相关的生物工程方法和研究证据。它讨论了由与CNS疾病相关的慢性坐姿所导致的深部组织中的独特生物力学状况,总体而言,本综述表明,在此类患者中避免界面压力高于32 mmHg不一定是一种“减压”措施。