Sugihara N, Matsuzaki M, Kato Y
Department of Internal Medicine, Ube First Hospital.
Nihon Ronen Igakkai Zasshi. 1991 Nov;28(6):829-36. doi: 10.3143/geriatrics.28.829.
A 76-year-old man was referred to our hospital with complaints of productive cough, dyspnea and peripheral cyanosis. The chest X-ray film indicated the pulmonary emphysema and acute bronchitis, but no abnormal intracardiac calcification. The electrocardiogram revealed a peaked P-wave, complete left bundle branch block, and ventricular premature contraction. Chest tomography demonstrated abnormal intracardiac calcium deposition in the right heart region. Two-dimensional echocardiography revealed the tricuspid annular calcification in the postero-lateral portion, showing a synchronous movement with tricuspid annular motion throughout the cardiac cycle. The size of calcification was 10 x 14 mm. The tricuspid valve showed no significant regurgitation. Left ventricular dilatation, associated with mild mitral regurgitation and impairment of systolic function (EF = 49%) was revealed by echocardiography. Serum examination revealed positive in Wassermann reaction. This case of tricuspid annular calcification might be caused by atherosclerotic degenerative change related to the aging process, or by an unknown mechanism related to pulmonary emphysema.
一名76岁男性因咳嗽、呼吸困难和外周发绀被转诊至我院。胸部X光片显示肺气肿和急性支气管炎,但心脏内无异常钙化。心电图显示P波高尖、完全性左束支传导阻滞和室性早搏。胸部断层扫描显示右心区域心脏内有异常钙沉积。二维超声心动图显示后外侧部分三尖瓣环钙化,在整个心动周期中与三尖瓣环运动同步。钙化大小为10×14毫米。三尖瓣无明显反流。超声心动图显示左心室扩张,伴有轻度二尖瓣反流和收缩功能受损(射血分数=49%)。血清检查显示瓦氏反应阳性。该例三尖瓣环钙化可能是由与衰老过程相关的动脉粥样硬化退行性改变引起,或者是由与肺气肿相关的未知机制引起。