相似文献
1
Beta-amyloid modulation of synaptic transmission and plasticity.
J Neurosci. 2007 Oct 31;27(44):11832-7. doi: 10.1523/JNEUROSCI.3478-07.2007.
2
Amyloid β: linking synaptic plasticity failure to memory disruption in Alzheimer's disease.
J Neurochem. 2012 Jan;120 Suppl 1(Suppl 1):140-148. doi: 10.1111/j.1471-4159.2011.07506.x. Epub 2011 Nov 28.
3
Amyloid-β induced signaling by cellular prion protein and Fyn kinase in Alzheimer disease.
Prion. 2013 Jan-Feb;7(1):37-41. doi: 10.4161/pri.22212. Epub 2012 Sep 17.
4
A 'danse macabre': tau and Fyn in STEP with amyloid beta to facilitate induction of synaptic depression and excitotoxicity.
Eur J Neurosci. 2013 Jun;37(12):1925-30. doi: 10.1111/ejn.12251.
5
Generation of aggregated beta-amyloid in the rat hippocampus impairs synaptic transmission and plasticity and causes memory deficits.
J Neurosci. 2001 Aug 1;21(15):5703-14. doi: 10.1523/JNEUROSCI.21-15-05703.2001.
6
Amyloid-β/Fyn-induced synaptic, network, and cognitive impairments depend on tau levels in multiple mouse models of Alzheimer's disease.
J Neurosci. 2011 Jan 12;31(2):700-11. doi: 10.1523/JNEUROSCI.4152-10.2011.
7
Potentiation of amyloid beta phagocytosis and amelioration of synaptic dysfunction upon FAAH deletion in a mouse model of Alzheimer's disease.
J Neuroinflammation. 2021 Sep 29;18(1):223. doi: 10.1186/s12974-021-02276-y.
8
REDD1 Is Involved in Amyloid β-Induced Synaptic Dysfunction and Memory Impairment.
Int J Mol Sci. 2020 Dec 13;21(24):9482. doi: 10.3390/ijms21249482.
9
Blocking the Interaction between EphB2 and ADDLs by a Small Peptide Rescues Impaired Synaptic Plasticity and Memory Deficits in a Mouse Model of Alzheimer's Disease.
J Neurosci. 2016 Nov 23;36(47):11959-11973. doi: 10.1523/JNEUROSCI.1327-16.2016.
10
Impairments of hippocampal synaptic plasticity induced by aggregated beta-amyloid (25-35) are dependent on stimulation-protocol and genetic background.
Exp Brain Res. 2007 Jun;179(4):621-30. doi: 10.1007/s00221-006-0819-6. Epub 2006 Dec 15.
引用本文的文献
1
Unlocking the Neuroprotective Potential of Silymarin: A Promising Ally in Safeguarding the Brain from Alzheimer's Disease and Other Neurological Disorders.
Mol Neurobiol. 2025 Jun;62(6):7975-7997. doi: 10.1007/s12035-024-04654-y. Epub 2025 Feb 17.
2
Advanced Immunolabeling Method for Optical Volumetric Imaging Reveals Dystrophic Neurites of Dopaminergic Neurons in Alzheimer's Disease Mouse Brain.
Mol Neurobiol. 2024 Jul;61(7):3976-3999. doi: 10.1007/s12035-023-03823-9. Epub 2023 Dec 4.
3
Extrasynaptic NMDA receptors in acute and chronic excitotoxicity: implications for preventive treatments of ischemic stroke and late-onset Alzheimer's disease.
Mol Neurodegener. 2023 Jul 3;18(1):43. doi: 10.1186/s13024-023-00636-1.
4
Amyloid Pathology in the Central Auditory Pathway of 5XFAD Mice Appears First in Auditory Cortex.
J Alzheimers Dis. 2022;89(4):1385-1402. doi: 10.3233/JAD-220538.
5
Bioactive human Alzheimer brain soluble Aβ: pathophysiology and therapeutic opportunities.
Mol Psychiatry. 2022 Aug;27(8):3182-3191. doi: 10.1038/s41380-022-01589-5. Epub 2022 Apr 28.
6
Association between Sleep, Alzheimer's, and Parkinson's Disease.
Biology (Basel). 2021 Nov 3;10(11):1127. doi: 10.3390/biology10111127.
7
The Implication of STEP in Synaptic Plasticity and Cognitive Impairments in Alzheimer's Disease and Other Neurological Disorders.
Front Cell Dev Biol. 2021 Jun 14;9:680118. doi: 10.3389/fcell.2021.680118. eCollection 2021.
8
Temporal Lobe Epilepsy and Alzheimer's Disease: From Preclinical to Clinical Evidence of a Strong Association.
J Alzheimers Dis Rep. 2021 Apr 6;5(1):243-261. doi: 10.3233/ADR-200286.
9
Application of expert system and LSTM in extracting index of synaptic plasticity.
Cogn Neurodyn. 2021 Apr;15(2):253-263. doi: 10.1007/s11571-020-09610-9. Epub 2020 Jun 29.
10
d-glutamate and Gut Microbiota in Alzheimer's Disease.
Int J Mol Sci. 2020 Apr 11;21(8):2676. doi: 10.3390/ijms21082676.
本文引用的文献
1
Aberrant excitatory neuronal activity and compensatory remodeling of inhibitory hippocampal circuits in mouse models of Alzheimer's disease.
Neuron. 2007 Sep 6;55(5):697-711. doi: 10.1016/j.neuron.2007.07.025.
2
Intracellular amyloid-beta in Alzheimer's disease.
Nat Rev Neurosci. 2007 Jul;8(7):499-509. doi: 10.1038/nrn2168.
3
Internalized antibodies to the Abeta domain of APP reduce neuronal Abeta and protect against synaptic alterations.
J Biol Chem. 2007 Jun 29;282(26):18895-906. doi: 10.1074/jbc.M700373200. Epub 2007 Apr 27.
4
Natural oligomers of the Alzheimer amyloid-beta protein induce reversible synapse loss by modulating an NMDA-type glutamate receptor-dependent signaling pathway.
J Neurosci. 2007 Mar 14;27(11):2866-75. doi: 10.1523/JNEUROSCI.4970-06.2007.
5
Abeta oligomer-induced aberrations in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alzheimer's disease.
J Neurosci. 2007 Jan 24;27(4):796-807. doi: 10.1523/JNEUROSCI.3501-06.2007.
6
Amyloid precursor protein overexpression depresses excitatory transmission through both presynaptic and postsynaptic mechanisms.
Proc Natl Acad Sci U S A. 2007 Jan 2;104(1):353-8. doi: 10.1073/pnas.0608807104. Epub 2006 Dec 21.
7
Beta-amyloid, oxidative stress and down syndrome.
Curr Alzheimer Res. 2006 Dec;3(5):521-8. doi: 10.2174/156720506779025305.
8
AMPAR removal underlies Abeta-induced synaptic depression and dendritic spine loss.
Neuron. 2006 Dec 7;52(5):831-43. doi: 10.1016/j.neuron.2006.10.035.
9
Intracellular Abeta and cognitive deficits precede beta-amyloid deposition in transgenic arcAbeta mice.
Neurobiol Aging. 2007 Sep;28(9):1297-306. doi: 10.1016/j.neurobiolaging.2006.06.019. Epub 2006 Jul 31.
10
Synapse formation and function is modulated by the amyloid precursor protein.
J Neurosci. 2006 Jul 5;26(27):7212-21. doi: 10.1523/JNEUROSCI.1450-06.2006.
Zotero 插件