Mori M, Takamori M
Neurology. 1976 Sep;26(9):882-7. doi: 10.1212/wnl.26.9.882.
In a 50-year-old man with hyperthyroidism and myasthenic weakness electrophysiologic phenomena similar to Eaton-Lambert syndrome were seen in classical myasthenia gravis. The orbicularis oculi showed an abnormally small muscle action potential in response to nerve stimulus and unusual facilitation of the response with activity. These signs are compatible with Eaton-Lambert syndrome, but the findings obtained from the adductor pollicis were typical of classical myasthenia. A possible common basis for hyperthyroidism and such an electrophysiologic complexity of transmission failure was considered as a pathogenic hypothesis.
在一名患有甲状腺功能亢进症和肌无力的50岁男性中，经典型重症肌无力出现了类似于伊顿 - 兰伯特综合征的电生理现象。眼轮匝肌对神经刺激的反应显示出异常小的肌肉动作电位，并且随着活动出现异常的反应易化现象。这些体征与伊顿 - 兰伯特综合征相符，但拇内收肌的检查结果是经典型重症肌无力的典型表现。甲状腺功能亢进症与这种电生理复杂的传递失败之间可能存在的共同基础被视为一种致病假说。
