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Neuropeptide Y-induced pressor responses in spontaneously hypertensive and Wistar-Kyoto rats antagonized by D-myo-inositol-1,2,6-triphosphate (PP56).

作者信息

Sun X Y, Edvinsson L, Hedner T

机构信息

Department of Pharmacology, University of Gothenburg, Sweden.

出版信息

J Hypertens Suppl. 1991 Dec;9(6):S344-5.

PMID:1818993
Abstract
摘要

相似文献

1
Neuropeptide Y-induced pressor responses in spontaneously hypertensive and Wistar-Kyoto rats antagonized by D-myo-inositol-1,2,6-triphosphate (PP56).D-肌醇-1,2,6-三磷酸（PP56）拮抗神经肽Y诱导的自发性高血压大鼠和Wistar-Kyoto大鼠的升压反应。
J Hypertens Suppl. 1991 Dec;9(6):S344-5.
2
Cardiovascular effects of alpha-trinositol in spontaneously hypertensive and normotensive Wistar-Kyoto rats.α-三肌醇对自发性高血压和正常血压的Wistar-Kyoto大鼠的心血管作用。
J Hypertens. 1993 Sep;11(9):935-43. doi: 10.1097/00004872-199309000-00007.
3
Effects of d-myo-inositol-1,2,6-trisphosphate on neuropeptide Y-induced potentiation of various vasoconstrictor agents in the rat.D-肌醇-1,2,6-三磷酸对大鼠中神经肽Y诱导的多种血管收缩剂增强作用的影响。
J Pharmacol Exp Ther. 1992 Jun;261(3):1147-52.
4
Role of endothelium on the effects of neuropeptide Y in mesenteric resistance arteries of spontaneously hypertensive and Wistar-Kyoto normotensive rats.内皮在神经肽Y对自发性高血压大鼠和Wistar-Kyoto正常血压大鼠肠系膜阻力动脉作用中的角色
J Pharmacol Exp Ther. 1991 Apr;257(1):276-81.
5
Enhanced slow-pressor response to angiotensin II in spontaneously hypertensive rats.自发性高血压大鼠对血管紧张素II的慢升压反应增强。
J Pharmacol Exp Ther. 1989 Dec;251(3):909-21.
6
Renovascular effects of sympathetic cotransmitters ATP and NPY are age-dependent in spontaneously hypertensive rats.交感神经共递质ATP和NPY对自发性高血压大鼠的肾血管作用具有年龄依赖性。
Cardiovasc Res. 2005 May 1;66(2):345-52. doi: 10.1016/j.cardiores.2004.12.005. Epub 2005 Jan 8.
7
Use of D-myo inositol 1,2,6 trisphosphate to inhibit contractile activity in rat ventricular cardiomyocytes induced by neuropeptide Y and other cardioactive peptides through phospholipase C.使用D-肌醇1,2,6-三磷酸通过磷脂酶C抑制神经肽Y和其他心血管活性肽诱导的大鼠心室心肌细胞的收缩活性。
Br J Pharmacol. 1997 Dec;122(8):1655-60. doi: 10.1038/sj.bjp.0701567.
8
Involvement of the vascular renin-angiotensin system in beta adrenergic receptor-mediated facilitation of vascular neurotransmission in spontaneously hypertensive rats.血管肾素-血管紧张素系统参与β肾上腺素能受体介导的自发性高血压大鼠血管神经传递促进作用。
J Pharmacol Exp Ther. 1984 Oct;231(1):23-32.
9
Sensitivity of pressor responses to central hypertonic saline is greatly enhanced even in pre-hypertensive spontaneously hypertensive rats.即使在高血压前期的自发性高血压大鼠中，加压反应对中枢高渗盐水的敏感性也会大大增强。
Neurosci Lett. 2006 May 22;399(3):255-8. doi: 10.1016/j.neulet.2006.02.002. Epub 2006 Feb 21.
10
D-myo-inositol-1,2,6-trisphosphate is a selective antagonist of neuropeptide Y-induced pressor responses in the pithed rat.D-肌醇-1,2,6-三磷酸是去脑大鼠中神经肽Y诱导的升压反应的选择性拮抗剂。
Eur J Pharmacol. 1991 Nov 12;204(3):281-6. doi: 10.1016/0014-2999(91)90853-i.

引用本文的文献

1
Effects of glibenclamide on the regional haemodynamic actions of alpha-trinositol and its influence on responses to vasodilators in conscious rats.格列本脲对α-三肌醇局部血流动力学作用的影响及其对清醒大鼠血管舒张剂反应的影响。
Br J Pharmacol. 1996 Feb;117(3):507-515. doi: 10.1111/j.1476-5381.1996.tb15219.x.
2
Effects of chronic infusions of alpha-trinositol on regional and cardiac haemodynamics in conscious rats.慢性输注α-三肌醇对清醒大鼠局部和心脏血流动力学的影响。
Br J Pharmacol. 1994 Sep;113(1):129-36. doi: 10.1111/j.1476-5381.1994.tb16184.x.