Katsuno Makoto, Kobayashi Shiro, Yokota Hiroyuki, Teramoto Akira
Department of Neurosurgery, Nippon Medical School, Chiba Hokuso Hospital, Inba-gun, Chiba, Japan.
Brain Nerve. 2008 Jan;60(1):89-91.
Two patients with primary oculomotor nerve palsy due to direct mild head injury are reported. They presented with internal ophthalmoplegia, dilated nonreactive pupils, and very mild disturbance in consciousness. Except for the persistent oculomotor nerve palsy, both the patients recovered fully within one week. Neither demonstrated a history that was suggestive of a cause for their oculomotor nerve palsy. Initial CT scans demonstrated localized subarachnoid hemorrhage around the brain stem. One of the patients had sustained a fracture of the anterior clinoid process. As the underlying pathophysiologic mechanism underlying the oculomotor nerve palsy we suspected mild injury to the pupillomotor fibers at the anterior petroclinoidal ligament and that of the pupillary fibers at the posterior petroclinoidal ligament. We speculate that these perforating fibers at the anterior petroclinoidal liqament acted as a fulcrum due to downward displacement of the brainstem at the time of impact.
报告了2例因轻度头部直接损伤导致原发性动眼神经麻痹的患者。他们表现为眼内肌麻痹、瞳孔散大且无反应,意识仅有非常轻微的障碍。除持续性动眼神经麻痹外,两名患者均在一周内完全康复。两人均无提示动眼神经麻痹病因的病史。最初的CT扫描显示脑干周围局限性蛛网膜下腔出血。其中一名患者前床突骨折。作为动眼神经麻痹的潜在病理生理机制,我们怀疑岩斜前韧带处的瞳孔运动纤维和岩斜后韧带处的瞳孔纤维受到轻度损伤。我们推测,由于撞击时脑干向下移位,岩斜前韧带处的这些穿通纤维起到了支点的作用。