Fantin A, Valenzuela R, Luco C, Vicuña X, Schweitzer M
Departamento de Neurología y Neurocirugía, Escuela de Medicina, Universidad Católica de Chile, Santiago.
Rev Med Chil. 1991 Jul;119(7):798-802.
A 15 year old patient developed ataxia 2 weeks after an upper respiratory infection. Absent reflexes, external ophthalmoplegia, bilateral ptosis, isochoric mydriasis and week photomotor reflexes (Bell's phenomenon) were noted. A slight increase in protein but not cell content of the CSF was observed. Miller-Fisher syndrome was diagnosed on clinical grounds and visual and auditory evoked potentials were explored and found to be normal. This is in complete agreement to the literature and indicates indemnity of the central nervous system in Miller-Fisher's syndrome. Peripheral nervous system abnormalities that have been identified by pathologic and radiologic studies may explain all of the alterations observed in this syndrome.
一名15岁患者在上呼吸道感染2周后出现共济失调。检查发现反射消失、外眼肌麻痹、双侧上睑下垂、等容性瞳孔散大以及微弱的光动反射（贝尔现象）。脑脊液蛋白略有增加，但细胞含量未增加。基于临床症状诊断为米勒-费希尔综合征，并对视觉和听觉诱发电位进行了检测，结果正常。这与文献完全相符，表明米勒-费希尔综合征中枢神经系统不受累。病理和放射学研究发现的周围神经系统异常可能解释该综合征中观察到的所有改变。
