Rao P R
Stroke Recovery Program, National Rehabilitation Hospital, Washington, D.C. 20010.
Clin Commun Disord. 1991 Spring;1(1):31-7.
R. L. was a 52-year-old man who was referred for an SLP consultation to determine the nature of his fluency disorder, whether or not treatment would be beneficial, and finally whether resumption of pre-trauma vocational status was feasible. The patient was involved in a motor vehicle accident with no resulting detectable trauma. However, shortly after the accident, R. L. developed a severe dysfluency that was later described as cortical stuttering. We reviewed the medical and rehabilitation work-up that attempted to determine whether the communication disorder was functional or organic in origin. Once the fluency disorder was determined to be caused by a suspected small, focal, hemispheric lesion, a five-month treatment program was undertaken that used a noval prosthetic approach to restore fluency. Once fluency was restored with the use of an artificial larynx, a residual anomia was detected and treated. The case of R. L. illustrates a stuttering that appeared to be caused by a combined neurogenic dyspraxic (vocal control), dysarthric (motor control), and dysnomic (word-finding) dysfluency. The literature on this issue was reviewed and the underlying mechanism of recovery was discussed.
R.L.是一名52岁男性,因言语语言病理学家(SLP)会诊前来,目的是确定其流畅性障碍的性质、治疗是否有益,以及最终恢复创伤前职业状态是否可行。患者遭遇了一场机动车事故,未造成可检测到的创伤。然而,事故发生后不久,R.L.出现了严重的言语不流畅,后来被描述为皮质性口吃。我们回顾了医学和康复检查,试图确定这种沟通障碍是功能性还是器质性原因。一旦确定流畅性障碍是由疑似小的、局灶性的半球病变引起,便开展了一个为期五个月的治疗项目,采用一种新型的修复方法来恢复流畅性。使用人工喉恢复流畅性后,检测并治疗了残留的命名障碍。R.L.的案例说明了一种口吃现象,它似乎是由神经源性言语运用障碍(发声控制)、构音障碍(运动控制)和命名性言语不流畅(找词困难)共同引起的。本文回顾了关于这个问题的文献,并讨论了恢复的潜在机制。