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基于人群的非裔美国婴儿猝死综合征队列中,致心律失常、易引发猝死的钠通道多态性S1103Y的过度表现。

Overrepresentation of the proarrhythmic, sudden death predisposing sodium channel polymorphism S1103Y in a population-based cohort of African-American sudden infant death syndrome.

作者信息

Van Norstrand David W, Tester David J, Ackerman Michael J

机构信息

Department of Molecular Pharmacology and Experimental Therapeutics, Mayo Clinic, Rochester, Minnesota 55905, USA.

出版信息

Heart Rhythm. 2008 May;5(5):712-5. doi: 10.1016/j.hrthm.2008.02.012. Epub 2008 Feb 16.

DOI:10.1016/j.hrthm.2008.02.012
PMID:18452875
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2430043/
Abstract

BACKGROUND

The S1103Y-SCN5A polymorphism has been implicated as a proarrhythmic, sudden death predisposing risk factor in African Americans, including one postmortem investigation of African-American infants with sudden infant death syndrome (SIDS).

OBJECTIVE

The purpose of this study was to assess whether the relatively African-American-specific common polymorphism S1103Y in the SCN5A-encoded cardiac sodium channel is overrepresented in SIDS among African Americans.

METHODS

Seventy-one cases from a population-based cohort of unexplained infant deaths among African Americans (37 females and 34 males, average age 3 +/- 2 months, age range birth to 11 months) were submitted to the Mayo Clinic Windland Smith Rice Sudden Death Genomics Laboratory for postmortem genetic testing. Polymerase chain reaction and a restriction digest assay were performed to genotype this cohort for S1103Y.

RESULTS

Targeted mutational analysis of exon 18 in SCN5A of the African-American SIDS cohort (n = 71) revealed the S1103Y polymorphism in 16 (22.5%) of 71 African-American cases of SIDS compared to 135 (11.6%) of 1,161 ostensibly healthy adult African Americans (P = .01).

CONCLUSION

This study provides an independent assessment of the prevalence of S1103Y-SCN5A among African-American infants with sudden, unexpected, unexplained death prior to their first birthday. Further scrutiny and quantification of the risk apparently associated with S1103Y appear warranted.

摘要

背景

S1103Y-SCN5A多态性被认为是非洲裔美国人发生心律失常和猝死的易感风险因素,包括一项对患有婴儿猝死综合征(SIDS)的非裔美国婴儿的尸检调查。

目的

本研究的目的是评估SCN5A编码的心脏钠通道中相对特定于非裔美国人的常见多态性S1103Y在非裔美国人SIDS中是否过度存在。

方法

来自一个基于人群的非裔美国人不明原因婴儿死亡队列的71例病例(37名女性和34名男性,平均年龄3±2个月,年龄范围从出生到11个月)被提交到梅奥诊所温德兰·史密斯·赖斯猝死基因组学实验室进行尸检基因检测。进行聚合酶链反应和限制性消化测定以对该队列进行S1103Y基因分型。

结果

对非裔美国人SIDS队列(n = 71)的SCN5A第18外显子进行靶向突变分析,发现71例非裔美国人SIDS病例中有16例(22.5%)存在S1103Y多态性,而在1161名表面健康的成年非裔美国人中有135例(11.6%)存在该多态性(P = .01)。

结论

本研究对1岁前突然、意外、不明原因死亡的非裔美国婴儿中S1103Y-SCN5A的患病率进行了独立评估。显然有必要对与S1103Y相关的风险进行进一步审查和量化。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3194/2430043/3d5e0cf10eb1/nihms48806f2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3194/2430043/8522960bbc90/nihms48806f1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3194/2430043/3d5e0cf10eb1/nihms48806f2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3194/2430043/8522960bbc90/nihms48806f1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3194/2430043/3d5e0cf10eb1/nihms48806f2.jpg

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