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[患有脓毒症的新生儿弥散性血管内凝血综合征过程中低凝血症紊乱的发展机制]

[Mechanism of the development of hypocoagulation disorders in the course of disseminated intravascular coagulation syndrome in newborn infants with pyo-septic diseases].

作者信息

Dmitriev V V

出版信息

Anesteziol Reanimatol. 1991 Mar-Apr(2):33-5.

PMID:1862984
Abstract

The development of hypocoagulation disturbances in sepsis of the newborn is mediated by the consumption of coagulation and vascular-platelet hemostasis factors. The formation of an insufficient clot in a number of patients may result in an increase of spontaneous fibrinolysis index, which does not correspond to the degree of decreased plasmin system activity due to its depletion.

摘要

新生儿脓毒症中低凝紊乱的发生是由凝血及血管-血小板止血因子的消耗介导的。在一些患者中,形成的凝血块不足可能导致自发纤溶指数升高,由于纤溶系统的消耗,该指数与纤溶系统活性降低的程度并不相符。

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