Firing first: compensatory changes in K+ channel knockout mice preserve excitability but not synaptic scaling.
作者信息
Hoffman Dax
机构信息
NIH, Laboratory of Cellular and Synaptic Neurophysiology, Rockville Pike 9000A, Bethesda, MD 20892, USA.
出版信息
J Physiol. 2008 Aug 15;586(16):3731-2. doi: 10.1113/jphysiol.2008.159335.
Abstract
摘要
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Deletion of Kv4.2 gene eliminates dendritic A-type K+ current and enhances induction of long-term potentiation in hippocampal CA1 pyramidal neurons.Kv4.2基因的缺失消除了海马CA1锥体神经元树突状A 型钾电流，并增强了长时程增强的诱导。
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