Terai C, Oryuh T, Kimura S, Mizuno K, Okada Y, Mimura K
Department of Traumatology and Emergency Medicine, National Defense Medical College, Saitama, Japan.
J Trauma. 1991 Aug;31(8):1165-8.
We examined the cardiovascular response of external leg counterpressure in healthy volunteers at 100 mm Hg compression pressure. To stimulate mild hypovolemia, measurements were made with the subjects in a 60 degrees head-up tilt position. Left ventricular end-diastolic volume (LVEDV) and cardiac output (CO) were calculated from two-dimensional echocardiography. Flow through the inferior vena cava (IVC) below the origin of the hepatic veins was determined by the Doppler ultrasound technique. The application of counterpressure significantly increased LVEDV, CO, and arterial blood pressure over that seen with tilting without the device. These responses were accompanied by a small but significant increase in IVC flow. We therefore concluded that external leg counterpressure transferred blood to the central circulation by compression of the venous capacitance vessels (an autotransfusion effect) in mild hypovolemia, but such an effect may not benefit patients in a hypovolemic shock state because of the small amount of translocated blood.
我们在100毫米汞柱的压缩压力下,对健康志愿者进行了下肢外部反压的心血管反应研究。为了模拟轻度血容量不足,在受试者处于头高位倾斜60度的姿势下进行测量。左心室舒张末期容积(LVEDV)和心输出量(CO)通过二维超声心动图计算得出。肝静脉起始部以下的下腔静脉(IVC)血流通过多普勒超声技术测定。与未使用该装置的倾斜状态相比,施加反压显著增加了LVEDV、CO和动脉血压。这些反应伴随着IVC血流的小幅但显著增加。因此,我们得出结论,在轻度血容量不足时,下肢外部反压通过压缩静脉容量血管将血液转移至中心循环(自体输血效应),但由于转移的血液量较少,这种效应可能对处于低血容量休克状态的患者无益处。