Ter-Nikogosian V A, Vartanian M K, Trchunian A A
Biofizika. 1991 Mar-Apr;36(2):281-5.
Bacteriophages P22 and dp8 cause the membrane potential depolarization for 10-30 mV, reversal rapid H+ influx into bacteria and K+ exit from S. typhimurium LT2, these effects depend on infection plural and are observed only in the presence of Ca+2 in the medium. delta psi depolarization and K+ efflux induced by phage dp8 are increased with the growth of Mg+2 concentration from 0 to 2 mM. Changes of delta pH and also Na+,Ca+2 concentrations are not observed. In the presence of glucose phage infection leads to changes in H(+)-K(+)-exchange. The phages P22 and dp8 adsorption on bacteria causes changes in the form or turn of the channels in S. typhimurium membrane.
噬菌体P22和dp8可使膜电位去极化10 - 30毫伏,迅速逆转氢离子流入细菌以及钾离子从鼠伤寒沙门氏菌LT2流出的过程,这些效应取决于感染复数,且仅在培养基中存在钙离子时才能观察到。噬菌体dp8诱导的Δψ去极化和钾离子外流会随着镁离子浓度从0增加到2毫摩尔而增强。未观察到ΔpH以及钠离子、钙离子浓度的变化。在有葡萄糖存在的情况下,噬菌体感染会导致H(+)-K(+)交换发生变化。噬菌体P22和dp8吸附在细菌上会导致鼠伤寒沙门氏菌膜中通道的形态或转向发生变化。