宿主对细菌通讯信号的拦截。
Host interception of bacterial communication signals.
作者信息
Horswill Alexander R, Nauseef William M
机构信息
Department of Microbiology, Roy J and Lucille A Carver College of Medicine, University of Iowa, Iowa City, IA 52242, USA.
出版信息
Cell Host Microbe. 2008 Dec 11;4(6):507-9. doi: 10.1016/j.chom.2008.11.003.
PMID:19064249
Abstract
Pathogenic Staphylococcus aureus employs a cell-to-cell communication system to control virulence factor expression in the host during infection. In this issue, Peterson et al. (2008) report that host lipoproteins can sequester the S. aureus communication signal and antagonize virulence factor expression and pathogenesis in a murine model of infection.
摘要
致病性金黄色葡萄球菌利用一种细胞间通讯系统来控制感染期间宿主中毒力因子的表达。在本期杂志中，彼得森等人（2008年）报道，宿主脂蛋白可以隔离金黄色葡萄球菌的通讯信号，并在小鼠感染模型中拮抗毒力因子的表达和发病机制。
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