Shevchenko T F, Khlevutin I S
Patol Fiziol Eksp Ter. 1991 Mar-Apr(2):47-9.
The entry of metabolites of the Aspergillus flavus mold into the gastrointestinal tract (GIT) with the food for one week causes inhibition of the activity of ATPase, cytochrome oxidase, and alkaline phosphatase; paranecrotic and morphologic changes in the tissue of the smooth muscles of the small intestine are encountered, which, evidently, underlies the disorders of intestinal digestion. The morphofunctional status of the small intestine is restored 5 days after the supply of mold metabolites into the GIT ceases.
黄曲霉霉菌的代谢产物随食物进入胃肠道一周会导致ATP酶、细胞色素氧化酶和碱性磷酸酶的活性受到抑制;小肠平滑肌组织出现准坏死和形态学变化,这显然是肠道消化紊乱的基础。在向胃肠道供应霉菌代谢产物停止5天后,小肠的形态功能状态得以恢复。