Ondar U N, Vu Ch Kh, Ezhova T A
Ontogenez. 2008 Nov-Dec;39(6):430-6.
A new deletion allele of the APETALA1 (AP1) gene encoding a type II MADS-box protein with the key role in the initiation of flowering and development of perianth organs has been identified in A. thaliana. The deletion of seven amino acids in the conserved region of the K domain in the ap1-20 mutant considerably delayed flowering and led to a less pronounced abnormality in the corolla development compared to the ap1-3 and apl-6 alleles with low and medium expression, respectively. At the same time, a considerable stamen reduction has been revealed in ap1-20 as distinct from ap1-3 and ap1-6 alleles. These data indicate that the K domain of AP1 can be crucial for the initiation of flowering and expression regulation of B-class genes controlling stamen development.
在拟南芥中发现了一种新的APETALA1（AP1）基因缺失等位基因，该基因编码一种II型MADS-box蛋白，在开花起始和花被器官发育中起关键作用。与分别具有低表达和中等表达的ap1-3和ap1-6等位基因相比，ap1-20突变体K结构域保守区域中七个氨基酸的缺失显著延迟了开花，并导致花冠发育异常不那么明显。同时，与ap1-3和ap1-6等位基因不同，ap1-20中出现了明显的雄蕊减少。这些数据表明，AP1的K结构域对于开花起始和控制雄蕊发育的B类基因的表达调控可能至关重要。
