Heldwein Ekaterina E
Department of Molecular Biology and Microbiology, Tufts University School of Medicine, 136 Harrison Avenue, Boston, MA 02111, USA.
Structure. 2009 Feb 13;17(2):147-9. doi: 10.1016/j.str.2009.01.003.
Interaction of viral protein gp42 with its receptor is the trigger for the entry of Epstein-Barr virus into B cells. The structure of gp42 reported by Kirschner etal. (2009) in this issue of Structure suggests a likely triggering mechanism substantially different from that of a related herpesvirus.
病毒蛋白gp42与其受体的相互作用是爱泼斯坦-巴尔病毒进入B细胞的触发因素。Kirschner等人（2009年）在本期《结构》杂志上报道的gp42结构表明，其触发机制可能与相关疱疹病毒的触发机制有很大不同。
