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Transcriptional control of endothelial cell development.
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Induction of hematopoietic and endothelial cell program orchestrated by ETS transcription factor ER71/ETV2.
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ETS transcription factors in hematopoietic stem cell development.
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Wnt signaling positively regulates endothelial cell fate specification in the Fli1a-positive progenitor population via Lef1.
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Foxs and Ets in the transcriptional regulation of endothelial cell differentiation and angiogenesis.
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Regulation of endothelial and hematopoietic development by the ETS transcription factor Etv2.
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Human single-cell atlas analysis reveals heterogeneous endothelial signaling.
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Vascular endothelial growth factor signaling in health and disease: from molecular mechanisms to therapeutic perspectives.
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Angiogenesis, signaling pathways, and animal models.
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Mapping the transcriptional and epigenetic landscape of organotypic endothelial diversity in the developing and adult mouse.
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Microenvironmental determinants of endothelial cell heterogeneity.
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Protocol for efficient generation of human artery and vein endothelial cells from pluripotent stem cells.
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本文引用的文献

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Nkx2-5 transactivates the Ets-related protein 71 gene and specifies an endothelial/endocardial fate in the developing embryo.
Proc Natl Acad Sci U S A. 2009 Jan 20;106(3):814-9. doi: 10.1073/pnas.0807583106. Epub 2009 Jan 7.
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Combinatorial regulation of endothelial gene expression by ets and forkhead transcription factors.
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Lymph sacs are not required for the initiation of lymph node formation.
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Sox18 induces development of the lymphatic vasculature in mice.
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Genome-wide analysis of the zebrafish ETS family identifies three genes required for hemangioblast differentiation or angiogenesis.
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Prox1 physically and functionally interacts with COUP-TFII to specify lymphatic endothelial cell fate.
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Endoglin expression in blood and endothelium is differentially regulated by modular assembly of the Ets/Gata hemangioblast code.
Blood. 2008 Dec 1;112(12):4512-22. doi: 10.1182/blood-2008-05-157560. Epub 2008 Sep 19.
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Identification of COUP-TFII orphan nuclear receptor as a retinoic acid-activated receptor.
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Fli1 acts at the top of the transcriptional network driving blood and endothelial development.
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