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Toward personalized, targeted therapy of gastrointestinal stromal tumor.

作者信息

Trent Jonathan C

机构信息

Department of Sarcoma Medical Oncology, Sarcoma Research Center, The University of Texas, M. D. Anderson Cancer Center, Houston, TX.

出版信息

Gastrointest Cancer Res. 2008 Sep;2(5):256-7.

PMID:19259311
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2632559/
Abstract
摘要

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本文引用的文献

1
Unlucky number 13? Differential effects of KIT exon 13 mutation in gastrointestinal stromal tumors.不吉利的数字13?KIT基因第13外显子突变在胃肠道间质瘤中的不同效应。
Mol Oncol. 2008 Aug;2(2):161-3. doi: 10.1016/j.molonc.2008.05.002. Epub 2008 May 10.
2
Optimizing imatinib mesylate treatment in gastrointestinal stromal tumors.优化甲磺酸伊马替尼在胃肠道间质瘤中的治疗
Gastrointest Cancer Res. 2008 Sep;2(5):245-50.
3
Relationship of imatinib-free plasma levels and target genotype with efficacy and tolerability.无伊马替尼血浆水平与目标基因型与疗效和耐受性的关系。
Br J Cancer. 2008 May 20;98(10):1633-40. doi: 10.1038/sj.bjc.6604355. Epub 2008 May 6.
4
Phase III randomized, intergroup trial assessing imatinib mesylate at two dose levels in patients with unresectable or metastatic gastrointestinal stromal tumors expressing the kit receptor tyrosine kinase: S0033.III期随机组间试验,评估甲磺酸伊马替尼在两个剂量水平对表达kit受体酪氨酸激酶的不可切除或转移性胃肠道间质瘤患者的疗效:S0033。
J Clin Oncol. 2008 Feb 1;26(4):626-32. doi: 10.1200/JCO.2007.13.4452.
5
Association of intratumoral vascular endothelial growth factor expression and clinical outcome for patients with gastrointestinal stromal tumors treated with imatinib mesylate.甲磺酸伊马替尼治疗的胃肠道间质瘤患者瘤内血管内皮生长因子表达与临床结局的相关性
Clin Cancer Res. 2007 Nov 15;13(22 Pt 1):6727-34. doi: 10.1158/1078-0432.CCR-07-0895.
6
KIT mutations and dose selection for imatinib in patients with advanced gastrointestinal stromal tumours.晚期胃肠道间质瘤患者的KIT突变与伊马替尼剂量选择
Eur J Cancer. 2006 May;42(8):1093-103. doi: 10.1016/j.ejca.2006.01.030. Epub 2006 Apr 18.
7
Progression-free survival in gastrointestinal stromal tumours with high-dose imatinib: randomised trial.高剂量伊马替尼治疗胃肠道间质瘤的无进展生存期:随机试验
Lancet. 2004;364(9440):1127-34. doi: 10.1016/S0140-6736(04)17098-0.
8
Use of c-KIT/PDGFRA mutational analysis to predict the clinical response to imatinib in patients with advanced gastrointestinal stromal tumours entered on phase I and II studies of the EORTC Soft Tissue and Bone Sarcoma Group.在欧洲癌症研究与治疗组织(EORTC)软组织和骨肉瘤小组的I期和II期研究中,使用c-KIT/PDGFRA突变分析来预测晚期胃肠道间质瘤患者对伊马替尼的临床反应。
Eur J Cancer. 2004 Mar;40(5):689-95. doi: 10.1016/j.ejca.2003.11.025.
9
Prognostic value of KIT mutation type, mitotic activity, and histologic subtype in gastrointestinal stromal tumors.KIT突变类型、有丝分裂活性和组织学亚型在胃肠道间质瘤中的预后价值。
J Clin Oncol. 2002 Sep 15;20(18):3898-905. doi: 10.1200/JCO.2002.03.095.