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The onset of fever: new insights into its mechanism.
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Preoptic norepinephrine mediates the febrile response of guinea pigs to lipopolysaccharide.
Am J Physiol Regul Integr Comp Physiol. 2007 Sep;293(3):R1135-43. doi: 10.1152/ajpregu.00067.2007. Epub 2007 Jun 20.
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Preoptic nitric oxide attenuates endotoxic fever in guinea pigs by inhibiting the POA release of norepinephrine.
Am J Physiol Regul Integr Comp Physiol. 2007 Sep;293(3):R1144-51. doi: 10.1152/ajpregu.00068.2007. Epub 2007 Jun 20.
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Kupffer cell-generated PGE2 triggers the febrile response of guinea pigs to intravenously injected LPS.
Am J Physiol Regul Integr Comp Physiol. 2006 May;290(5):R1262-70. doi: 10.1152/ajpregu.00724.2005. Epub 2006 Jan 12.
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Putative antihyperpyretic factor induced by LPS in spleen of guinea pigs.
Am J Physiol Regul Integr Comp Physiol. 2005 Sep;289(3):R680-7. doi: 10.1152/ajpregu.00022.2005. Epub 2005 May 26.
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LPS-activated complement, not LPS per se, triggers the early release of PGE2 by Kupffer cells.
Am J Physiol Regul Integr Comp Physiol. 2005 Aug;289(2):R332-R339. doi: 10.1152/ajpregu.00567.2004. Epub 2005 Mar 31.
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Complement component c5a is integral to the febrile response of mice to lipopolysaccharide.
Neuroimmunomodulation. 2005;12(2):67-80. doi: 10.1159/000083578.
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Fever onset is linked to the appearance of lipopolysaccharide in the liver.
J Endotoxin Res. 2004;10(1):39-53. doi: 10.1179/096805104225003825.
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Signaling the brain in systemic inflammation: the role of complement.
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