Rhind Nicholas
Department of Biochemistry and Molecular Pharmacology, University of Massachusetts Medical School, 364 Plantation Street, Worcester, MA 01605, USA.
Mol Cell. 2009 Mar 27;33(6):672-4. doi: 10.1016/j.molcel.2009.03.004.
In a recent issue of Molecular Cell, Shiotani and Zou (2009) elucidate the biochemical mechanism underlying sequential ATM and ATR activation at DNA double-strand breaks, demonstrating that resection transforms ATM substrates into ATR substrates.
在最近一期的《分子细胞》杂志上，盐谷和邹（2009年）阐明了DNA双链断裂处ATM和ATR顺序激活背后的生化机制，证明了切除作用将ATM底物转化为ATR底物。
