Park Chul
Seoul, Korea From The Seoul Center for Developmental Ear Anomalies and the Department of Plastic and Reconstructive Surgery, Korea University Anam Hospital.
Plast Reconstr Surg. 2009 Apr;123(4):1302-1312. doi: 10.1097/PRS.0b013e31819e2679.
During treatment of upper auricular malformations, the author found that patients with cryptotia and patients with solitary helical and/or antihelical adhesion malformations showed the same anatomical finding of cartilage adhesion. The author defined them together as upper auricular adhesion malformations.
Between March of 1992 and March of 2006, 194 upper auricular adhesion malformations were corrected in 137 patients. All of these cases were retrospectively studied and classified. Of these, 92 malformations in 68 recent patients were corrected with new surgical methods (these were followed up for more than 6 months).
The group of solitary helical and/or antihelical cartilage malformation patients was classified as group I and the cryptotia group as group II. These two groups were subdivided according to features of cartilage adhesion and classified into seven subgroups. Thirty-two malformations were classified as belonging to group I and 162 malformations to group II. There were 61 patients with bilateral upper auricular adhesion malformations. Nineteen patients (31 percent of the patients with bilateral malformations) showed malformations belonging to both groups I and II on both ears. On postoperative observation in patients corrected with new methods, it was noticed that the following unfavorable results had occurred in 18 upper auricular adhesion malformation cases (20 percent): venous congestion or partial skin necrosis of used flaps, "pinched antitragus," low-set upper auricle, hypertrophic scars, and baldness.
The new consideration for, and the singling out of, upper auricular adhesion malformation can lead to better understanding of the groups of upper auricular malformations to which it belongs, the decision for treatment, and, possibly, clarification of the pathophysiology in the future.
在治疗耳廓上部畸形时,作者发现隐耳患者以及单独的耳轮和/或对耳轮粘连畸形患者表现出相同的软骨粘连解剖学特征。作者将它们统称为耳廓上部粘连畸形。
1992年3月至2006年3月期间,对137例患者的194例耳廓上部粘连畸形进行了矫正。对所有这些病例进行了回顾性研究和分类。其中,对68例近期患者的92例畸形采用新的手术方法进行了矫正(这些患者随访时间超过6个月)。
单独的耳轮和/或对耳轮软骨畸形患者组归为I组,隐耳组归为II组。根据软骨粘连特征将这两组再细分,分为七个亚组。32例畸形归为I组,162例畸形归为II组。有61例患者为双侧耳廓上部粘连畸形。19例患者(占双侧畸形患者的31%)双耳畸形同时属于I组和II组。在采用新方法矫正的患者术后观察中,发现18例耳廓上部粘连畸形病例(20%)出现了以下不良结果:所用皮瓣静脉淤血或部分皮肤坏死、“对耳屏受压”、耳廓上部低位、瘢痕增生和秃发。
对耳廓上部粘连畸形的新认识和单独划分,有助于更好地了解其所属的耳廓上部畸形组、治疗决策,并可能在未来阐明其病理生理学。