VEGF inhibition and metastasis: possible implications for antiangiogenic therapy.

作者信息

English Bevin C, Price Douglas K, Figg William D

机构信息

Molecular Pharmacology Section, Medical Oncology Branch, National Cancer Institute, NIH, Bethesda, MD 20892, USA.

出版信息

Cancer Biol Ther. 2009 Jul;8(13):1214-25. doi: 10.4161/cbt.8.13.8918. Epub 2009 Jul 4.

DOI:10.4161/cbt.8.13.8918

PMID:19483467

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2802271/

Abstract

摘要

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本文引用的文献

[1]

Accelerated metastasis after short-term treatment with a potent inhibitor of tumor angiogenesis.

Cancer Cell. 2009-3-3

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Antiangiogenic therapy elicits malignant progression of tumors to increased local invasion and distant metastasis.

Cancer Cell. 2009-3-3

[3]

Tumor angiogenesis.

N Engl J Med. 2008-5-8

[4]

Angiogenesis inhibitors and hypoxia.

Nat Med. 2003-9

[5]

Hypoxia promotes invasive growth by transcriptional activation of the met protooncogene.

Cancer Cell. 2003-4

[6]

Anti-VEGF antibody treatment of glioblastoma prolongs survival but results in increased vascular cooption.

Neoplasia. 2000

[7]

Effects of PTK787/ZK 222584, a specific inhibitor of vascular endothelial growth factor receptor tyrosine kinases, on primary tumor, metastasis, vessel density, and blood flow in a murine renal cell carcinoma model.

Cancer Res. 2000-9-1

[8]

Antiangiogenic therapy targeting the tyrosine kinase receptor for vascular endothelial growth factor receptor inhibits the growth of colon cancer liver metastasis and induces tumor and endothelial cell apoptosis.

Cancer Res. 1999-11-1

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VEGF inhibition and metastasis: possible implications for antiangiogenic therapy.

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