糖尿病胚胎病的生化基础。
Biochemical basis of the diabetic embryopathy.
作者信息
Goldman A S, Goto M P
机构信息
Center for Craniofacial Anomalies, University of Illinois, College of Medicine, Chicago 60612.
出版信息
Isr J Med Sci. 1991 Aug-Sep;27(8-9):469-77.
PMID:1960043
Abstract
Signal transduction, phosphatidylinositol turnover and the arachidonic acid cascade appear to be a final common pathway involved in the differentiation and development of the early embryo and several embryonic organ structures. Disruption of this biochemical pathway by various agents may account for the occurrence of the diabetic embryopathy.
摘要
信号转导、磷脂酰肌醇代谢及花生四烯酸级联反应似乎是参与早期胚胎及多个胚胎器官结构分化与发育的最终共同途径。各种因素对这一生物化学途径的破坏可能是糖尿病性胚胎病发生的原因。
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