"Ex vivo" binding of 35S-TBPS as a tool to study the pharmacology of GABAA receptors.
作者信息
Concas A, Sanna E, Serra M, Mascia M P, Santoro V, Biggio G
机构信息
Department of Experimental Biology, University of Cagliari, Italy.
出版信息
Adv Biochem Psychopharmacol. 1990;46:89-108.
PMID:1963271
Abstract
摘要
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"Ex vivo" binding of 35S-TBPS as a tool to study the pharmacology of GABAA receptors.作为研究GABAA受体药理学工具的35S-TBPS的“体外”结合
Adv Biochem Psychopharmacol. 1990;46:89-108.
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Isoniazid, an inhibitor of GABAergic transmission, enhances [35S]TBPS binding in rat cerebral cortex.异烟肼，一种γ-氨基丁酸能传递的抑制剂，可增强大鼠大脑皮层中[35S]TBPS的结合。
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Modulation of 35S-TBPS binding by GABAergic drugs in the cerebral cortex of newborn and adult rats.新生和成年大鼠大脑皮层中γ-氨基丁酸能药物对35S-TBPS结合的调节作用。
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Foot-shock stress and anxiogenic beta-carbolines increase t-[35S]butylbicyclophosphorothionate binding in the rat cerebral cortex, an effect opposite to anxiolytics and gamma-aminobutyric acid mimetics.足部电击应激和致焦虑的β-咔啉会增加大鼠大脑皮层中t-[35S]丁基双环磷硫代酸盐的结合，这一效应与抗焦虑药和γ-氨基丁酸模拟物相反。
J Neurochem. 1988 Dec;51(6):1868-76. doi: 10.1111/j.1471-4159.1988.tb01170.x.
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Foot-shock stress enhances the increase of [35S]TBPS binding in the rat cerebral cortex and the convulsions induced by isoniazid.足部电击应激增强了大鼠大脑皮层中[35S]TBPS结合的增加以及异烟肼诱导的惊厥。
Neurochem Res. 1991 Jan;16(1):17-22. doi: 10.1007/BF00965822.
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Changes of 36Cl- flux and 35S-TBPS binding induced by stress and gabaergic drugs.
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The effect of CNS depressants on GABA receptor chloride ionophore complexes.
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Functional correlation between allopregnanolone and [35S]-TBPS binding in the brain of rats exposed to isoniazid, pentylenetetrazol or stress.异烟肼、戊四氮或应激暴露大鼠大脑中别孕烯醇酮与[35S]-TBPS结合之间的功能相关性。
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