Smith Susan
Department of Pathology, Kimmel Center for Biology and Medicine of the Skirball Institute, New York University School of Medicine, New York, 10016, USA.
Mol Cell. 2009 Aug 14;35(3):256-8. doi: 10.1016/j.molcel.2009.07.011.
In this issue of Molecular Cell, Atanassov et al. (2009) show that the GCN5-containing SAGA complex regulates telomere function via deubiquitination and stabilization of the telomere repeat binding factor TRF1.
在本期《分子细胞》杂志中，阿塔纳索夫等人（2009年）表明，含有GCN5的SAGA复合物通过去泛素化和稳定端粒重复序列结合因子TRF1来调节端粒功能。
