Fitoussi Frank, Ilharreborde Brice, Guerin Florent, Souchet Philippe, Penneçot Georges F, Mazda Keyvan
Robert Debré Hospital, AP-HP, Paris and Denis Diderot University, Paris 7, France,
J Child Orthop. 2009 Oct;3(5):339-43. doi: 10.1007/s11832-009-0200-y. Epub 2009 Aug 22.
The development of claw toe deformity following fracture of the tibia in children has not been described in our review of the literature. We report on the management of the acquired claw toe deformity after tibia fracture in five children.
We report on five patients, between 5 and 15 years of age, who developed clawing of the hallux following a fracture of the tibia. In two patients, the lesser toes were involved. On examination, when the ankle was passively plantar flexed, a flexion contracture of the interphalangeal joint of the hallux became fully flexible. When the ankle was dorsiflexed, the clawing became more obvious and fixed. A magnetic resonance imaging (MRI) study in two cases demonstrated fibrosis under or just proximal to the tarsal tunnel.
The contractures were relieved by performing a tenolysis proximal to the medial malleolus. The operative findings demonstrated that the etiology could be possibly associated with a localized subclinical compartment syndrome.
We described five patients with a claw toe deformity following a tibia fracture associated with adhesions of the flexor hallucis longus (FHL) and flexor digitorum longus (FDL) muscles to the surrounding structures under or just proximal to the flexor retinaculum. It is the authors' opinion that this condition may be related to a subclinical compartment syndrome localized in the distal part of the deep posterior compartment. Soft-tissue release without tendon lengthening allowed recovery in all patients.
我们在文献回顾中未发现有关儿童胫骨骨折后爪形趾畸形发展情况的描述。我们报告了5例儿童胫骨骨折后获得性爪形趾畸形的治疗情况。
我们报告了5例年龄在5至15岁之间的患者,他们在胫骨骨折后出现了拇趾爪形畸形。其中2例患者的小趾也受累。检查时,当踝关节被动跖屈时,拇趾指间关节的屈曲挛缩变得完全可活动。当踝关节背屈时,爪形畸形变得更加明显且固定。2例患者的磁共振成像(MRI)研究显示跗管下方或其近端存在纤维化。
通过在内踝近端进行肌腱松解术,挛缩得以缓解。手术结果表明,病因可能与局部亚临床骨筋膜室综合征有关。
我们描述了5例胫骨骨折后出现爪形趾畸形的患者,其病因是拇长屈肌(FHL)和趾长屈肌(FDL)在屈肌支持带下方或其近端与周围结构粘连。作者认为这种情况可能与深后骨筋膜室远端局部的亚临床骨筋膜室综合征有关。所有患者在未进行肌腱延长的情况下进行软组织松解后均得以康复。