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Increased AS160 phosphorylation, but not TBC1D1 phosphorylation, with increased postexercise insulin sensitivity in rat skeletal muscle.
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Sustained AS160 and TBC1D1 phosphorylations in human skeletal muscle 30 min after a single bout of exercise.
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In vivo exercise followed by in vitro contraction additively elevates subsequent insulin-stimulated glucose transport by rat skeletal muscle.
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Contraction-stimulated glucose transport in rat skeletal muscle is sustained despite reversal of increased PAS-phosphorylation of AS160 and TBC1D1.
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引用本文的文献
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AS160 expression, but not AS160 Serine-588, Threonine-642, and Serine-704 phosphorylation, is essential for elevated insulin-stimulated glucose uptake by skeletal muscle from female rats after acute exercise.
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Akt substrate of 160 kDa is essential for the calorie restriction-induced increase in insulin-stimulated glucose uptake by skeletal muscle of female rats.
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Exercise effects on γ3-AMPK activity, Akt substrate of 160 kDa phosphorylation, and glucose uptake in muscle of normal and insulin-resistant female rats.
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Exercise-Induced Improvement in Insulin-Stimulated Glucose Uptake by Rat Skeletal Muscle Is Absent in Male AS160-Knockout Rats, Partially Restored by Muscle Expression of Phosphomutated AS160, and Fully Restored by Muscle Expression of Wild-Type AS160.
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The combined effect of magnetic and electric fields using on/off infrared light on the blood sugar level and the diameter of Langerhans islets of diabetic mice.
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TBC1D1 interacting proteins, VPS13A and VPS13C, regulate GLUT4 homeostasis in C2C12 myotubes.
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Noninvasive Continuous Glucose Monitoring Using a Multisensor-Based Glucometer and Time Series Analysis.
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Roux-en-Y gastric bypass surgery enhances contraction-mediated glucose metabolism in primary human myotubes.
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本文引用的文献
1
Increased AS160 phosphorylation, but not TBC1D1 phosphorylation, with increased postexercise insulin sensitivity in rat skeletal muscle.
Am J Physiol Endocrinol Metab. 2009 Jul;297(1):E242-51. doi: 10.1152/ajpendo.00194.2009. Epub 2009 May 12.
2
Potential role of TBC1D4 in enhanced post-exercise insulin action in human skeletal muscle.
Diabetologia. 2009 May;52(5):891-900. doi: 10.1007/s00125-009-1294-y. Epub 2009 Feb 28.
3
Rapid reversal of insulin-stimulated AS160 phosphorylation in rat skeletal muscle after insulin exposure.
Physiol Res. 2010;59(1):71-78. doi: 10.33549/physiolres.931707. Epub 2009 Feb 27.
4
Inhibition of contraction-stimulated AMP-activated protein kinase inhibits contraction-stimulated increases in PAS-TBC1D1 and glucose transport without altering PAS-AS160 in rat skeletal muscle.
Diabetes. 2009 May;58(5):1096-104. doi: 10.2337/db08-1477. Epub 2009 Feb 10.
5
A myosin II ATPase inhibitor reduces force production, glucose transport, and phosphorylation of AMPK and TBC1D1 in electrically stimulated rat skeletal muscle.
Am J Physiol Endocrinol Metab. 2009 May;296(5):E993-E1002. doi: 10.1152/ajpendo.91003.2008. Epub 2009 Feb 3.
6
Resistance exercise increases human skeletal muscle AS160/TBC1D4 phosphorylation in association with enhanced leg glucose uptake during postexercise recovery.
J Appl Physiol (1985). 2008 Dec;105(6):1967-74. doi: 10.1152/japplphysiol.90562.2008. Epub 2008 Oct 9.
7
Contraction-stimulated glucose transport in rat skeletal muscle is sustained despite reversal of increased PAS-phosphorylation of AS160 and TBC1D1.
J Appl Physiol (1985). 2008 Dec;105(6):1788-95. doi: 10.1152/japplphysiol.90838.2008. Epub 2008 Sep 25.
8
Muscle cells engage Rab8A and myosin Vb in insulin-dependent GLUT4 translocation.
Am J Physiol Cell Physiol. 2008 Oct;295(4):C1016-25. doi: 10.1152/ajpcell.00277.2008. Epub 2008 Aug 13.
9
Emerging role for AS160/TBC1D4 and TBC1D1 in the regulation of GLUT4 traffic.
Am J Physiol Endocrinol Metab. 2008 Jul;295(1):E29-37. doi: 10.1152/ajpendo.90331.2008. Epub 2008 May 13.
10
Discovery of TBC1D1 as an insulin-, AICAR-, and contraction-stimulated signaling nexus in mouse skeletal muscle.
J Biol Chem. 2008 Apr 11;283(15):9787-96. doi: 10.1074/jbc.M708839200. Epub 2008 Feb 13.
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