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氯磺丙脲对蟾蜍膀胱中血管加压素刺激的前列腺素E生物合成的抑制作用。增强血管加压素刺激的水流动的机制。

Inhibition of vasopressin-stimulated prostaglandin E biosynthesis by chlorpropamide in the toad urinary bladder. Mechanism of enhancement of vasopressin-stimulated water flow.

作者信息

Zusman R M, Keiser H R, Handler J S

出版信息

J Clin Invest. 1977 Dec;60(6):1348-53. doi: 10.1172/JCI108894.

DOI:10.1172/JCI108894

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC372491/

Abstract

Chlorpropamide is known to enhance the water permeability response of the toad urinary bladder to vasopressin and to theophylline. In other studies, we have shown that prostaglandin E synthesis by the toad bladder inhibits the water permeability response to arginine vasopressin and to theophylline. In this study, the effect of chlorpropamide on vasopressin-, theophylline-, and cyclic AMP-stimulated water flow and on prostaglandin E biosynthesis was investigated in the toad urinary bladder in vitro. Chlorpropamide inhibited prostaglandin E biosynthesis during vasopressin-, theophylline- and cyclic AMP-stimulated water flow. Tolbutamide and glyburide, two other sulfonylurea compounds, also enhanced vasopressin-stimulated water flow and inhibited vasopressin-stimulated prostaglandin E biosynthesis. We conclude that the mechanism of enhancement on vasopressin-stimulated water flow by the sulfonylureas is the inhibition of prostaglandin E biosynthesis.

摘要

已知氯磺丙脲可增强蟾蜍膀胱对血管加压素和茶碱的水通透性反应。在其他研究中，我们已表明蟾蜍膀胱合成的前列腺素E可抑制对精氨酸血管加压素和茶碱的水通透性反应。在本研究中，在体外蟾蜍膀胱中研究了氯磺丙脲对血管加压素、茶碱和环磷酸腺苷刺激的水流动以及对前列腺素E生物合成的影响。在血管加压素、茶碱和环磷酸腺苷刺激水流动期间，氯磺丙脲抑制前列腺素E的生物合成。另外两种磺脲类化合物甲苯磺丁脲和格列本脲也增强血管加压素刺激的水流动并抑制血管加压素刺激的前列腺素E生物合成。我们得出结论，磺脲类药物增强血管加压素刺激的水流动的机制是抑制前列腺素E的生物合成。