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潜艇从600至1000英尺海水深度逃生期间的氮麻醉和氧中毒估计值。

Estimates of N2 narcosis and O2 toxicity during submarine escapes from 600 to 1,000 fsw.

作者信息

Connor Christopher W, Ferrigno Massimo

机构信息

Department of Anesthesiology, Boston Medical Center, Boston, Massachusetts, USA.

出版信息

Undersea Hyperb Med. 2009 Jul-Aug;36(4):237-45.

Abstract

The U.S. Navy recommends submarine escape for depths down to 600 fsw, with deeper escapes entailing the risks of decompression sickness, nitrogen (N2) narcosis and CNS oxygen (O2) toxicity. However, the escape equipment, including the submarine escape and immersion equipment and the escape trunk, could probably function even at 1,000 fsw. Here we report a theoretical analysis of the risks of both N2 narcosis and CNS O2 toxicity for different escape profiles from 600 to 1,000 fsw. The effect of N2 narcosis, calculated as a function of N2 pressure in the brain using Gas Man software, was expressed as equivalent narcosis depth (END), corresponding to the depth at which the same pressure of N2 would be produced in the brain after five minutes of scuba diving with air. The risk of O2-induced convulsions was estimated using the model developed by Arieli et al. Different dwell times (DTs) at maximal pressure in the escape trunk (from 0 to 60 s) and lungs-to-brain circulation times (10 to 30 s) were included in our analysis. When DT in the escape trunk is very short (e.g., 10 s), the risk of either incapacitating N2 narcosis and/or O2-induced convulsions occurring in the trunk is low, even during escapes from 1,000 fsw.

摘要

美国海军建议在深度达600英尺海水(fsw)的情况下进行潜艇逃生,而更深深度的逃生则存在减压病、氮气(N₂)麻醉和中枢神经系统氧气(O₂)中毒的风险。然而,包括潜艇逃生和浸水设备以及逃生舱在内的逃生装备甚至在1000 fsw深度时可能仍能发挥作用。在此,我们报告了对从600至1000 fsw不同逃生剖面的N₂麻醉和中枢神经系统O₂中毒风险的理论分析。使用气体人软件计算得出的N₂麻醉效应作为大脑中N₂压力的函数,以等效麻醉深度(END)表示,相当于在使用空气进行水肺潜水五分钟后大脑中产生相同压力N₂的深度。使用阿列利等人开发的模型估算了O₂诱发惊厥的风险。我们的分析纳入了在逃生舱中最大压力下的不同停留时间(DT,从0至60秒)以及肺到脑的循环时间(10至30秒)。当逃生舱中的DT非常短(例如10秒)时,即使在从1000 fsw深度逃生期间,在舱内发生使人丧失能力的N₂麻醉和/或O₂诱发惊厥的风险也很低。

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