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兔实验性脊柱侧凸的产生机制。

Mechanism of production of experimental scoliosis in rabbits.

作者信息

Pal G P, Bhatt R H, Patel V S

机构信息

Department of Anatomy, Government Medical College, Surat, India.

出版信息

Spine (Phila Pa 1976). 1991 Feb;16(2):137-42.

PMID:2011768
Abstract

The hypothesis for the mechanism of production of scoliosis, advanced on the basis of morphometric and morphologic studies in human thoracic skeleton, finds further support in the results of an experimental study in rabbits described herein. The removal of transverse processes alone resulted in the production of scoliosis and lordosis. When both transverse processes and facet joints were removed, scoliosis developed rapidly and was considerable. When paraspinal muscles were incised on one side only, however, this resulted in the development of lordosis alone. The convexity of the curve was always toward the operated side, and the apex was almost always at the lowest operated segment. Lordosis was limited to within the operated area. The results of the experiment indicate that scoliosis resulted because of the asymmetry in load transmission through the ribs to the vertebral column in rabbits.

摘要

基于对人类胸廓骨骼的形态测量和形态学研究提出的脊柱侧弯产生机制假说,在本文所述的兔子实验研究结果中得到了进一步支持。仅切除横突会导致脊柱侧弯和脊柱前凸的产生。当横突和小关节都被切除时,脊柱侧弯迅速发展且程度严重。然而,仅在一侧切开椎旁肌肉,只会导致脊柱前凸的发展。曲线的凸侧总是朝向手术侧,顶点几乎总是在最低的手术节段。脊柱前凸仅限于手术区域内。实验结果表明,兔子脊柱侧弯是由于通过肋骨传递到脊柱的负荷不对称所致。

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Mechanism of production of experimental scoliosis in rabbits.兔实验性脊柱侧凸的产生机制。
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