相似文献
1
Ubiquitination and degradation of the inhibitors of NF-kappaB.
Cold Spring Harb Perspect Biol. 2010 Feb;2(2):a000166. doi: 10.1101/cshperspect.a000166.
2
Regulation of NF-κB by ubiquitination and degradation of the IκBs.
Immunol Rev. 2012 Mar;246(1):77-94. doi: 10.1111/j.1600-065X.2012.01098.x.
3
Hydrogen peroxide prolongs nuclear localization of NF-kappaB in activated cells by suppressing negative regulatory mechanisms.
J Biol Chem. 2008 Jul 4;283(27):18582-90. doi: 10.1074/jbc.M801312200. Epub 2008 May 12.
4
Nuclear ubiquitin ligases, NF-kappaB degradation, and the control of inflammation.
Sci Signal. 2008 Jan 8;1(1):pe1. doi: 10.1126/stke.11pe1.
5
Nuclear IKKbeta is an adaptor protein for IkappaBalpha ubiquitination and degradation in UV-induced NF-kappaB activation.
Mol Cell. 2010 Aug 27;39(4):570-82. doi: 10.1016/j.molcel.2010.07.030.
6
Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity.
Annu Rev Immunol. 2000;18:621-63. doi: 10.1146/annurev.immunol.18.1.621.
7
PDLIM2-mediated termination of transcription factor NF-kappaB activation by intranuclear sequestration and degradation of the p65 subunit.
Nat Immunol. 2007 Jun;8(6):584-91. doi: 10.1038/ni1464. Epub 2007 Apr 29.
8
IL-17 receptor signaling: ubiquitin gets in on the act.
Sci Signal. 2009 Oct 13;2(92):pe64. doi: 10.1126/scisignal.292pe64.
9
COMMD1 promotes the ubiquitination of NF-kappaB subunits through a cullin-containing ubiquitin ligase.
EMBO J. 2007 Jan 24;26(2):436-47. doi: 10.1038/sj.emboj.7601489. Epub 2006 Dec 21.
10
Nuclear retention of IkappaBalpha protects it from signal-induced degradation and inhibits nuclear factor kappaB transcriptional activation.
J Biol Chem. 1999 Mar 26;274(13):9108-15. doi: 10.1074/jbc.274.13.9108.
引用本文的文献
1
Multiomics profiles of genome-wide alterations in H3K27ac in different lung lobes after acute graft--host disease with MSCs treatment.
Front Immunol. 2025 May 15;16:1570916. doi: 10.3389/fimmu.2025.1570916. eCollection 2025.
2
Investigating Natural Product Inhibitors of IKKα: Insights from Integrative In Silico and Experimental Validation.
Molecules. 2025 May 2;30(9):2025. doi: 10.3390/molecules30092025.
3
TLR4 endocytosis and endosomal TLR4 signaling are distinct and independent outcomes of TLR4 activation.
EMBO Rep. 2025 Apr 9. doi: 10.1038/s44319-025-00444-2.
4
Ubiquitination of Immune System and Cancer Therapy.
Adv Exp Med Biol. 2024;1466:35-45. doi: 10.1007/978-981-97-7288-9_3.
5
E3 ubiquitin ligase gene modulates TNF-induced cell death pathways and promotes aberrant proliferation in rheumatoid arthritis fibroblast-like synoviocytes.
Front Immunol. 2024 Sep 5;15:1433898. doi: 10.3389/fimmu.2024.1433898. eCollection 2024.
6
Potential Application of Plant-Derived Compounds in Multiple Sclerosis Management.
Nutrients. 2024 Sep 5;16(17):2996. doi: 10.3390/nu16172996.
7
TRAF3 loss-of-function reveals the noncanonical NF-κB pathway as a therapeutic target in diffuse large B cell lymphoma.
Proc Natl Acad Sci U S A. 2024 Apr 30;121(18):e2320421121. doi: 10.1073/pnas.2320421121. Epub 2024 Apr 25.
8
Activity-based profiling of cullin-RING E3 networks by conformation-specific probes.
Nat Chem Biol. 2023 Dec;19(12):1513-1523. doi: 10.1038/s41589-023-01392-5. Epub 2023 Aug 31.
9
HSPB1 facilitates chemoresistance through inhibiting ferroptotic cancer cell death and regulating NF-κB signaling pathway in breast cancer.
Cell Death Dis. 2023 Jul 15;14(7):434. doi: 10.1038/s41419-023-05972-0.
10
Degron Pathways and Leishmaniasis: Debating Potential Roles of spp. Proteases Activity on Guiding Hosts Immune Response and Their Relevance to the Development of Vaccines.
Vaccines (Basel). 2023 May 23;11(6):1015. doi: 10.3390/vaccines11061015.
本文引用的文献
1
An inhibitor of NEDD8-activating enzyme as a new approach to treat cancer.
Nature. 2009 Apr 9;458(7239):732-6. doi: 10.1038/nature07884.
2
Modification by single ubiquitin moieties rather than polyubiquitination is sufficient for proteasomal processing of the p105 NF-kappaB precursor.
Mol Cell. 2009 Feb 27;33(4):496-504. doi: 10.1016/j.molcel.2009.01.023.
3
Proteolysis of NF-kappaB1 p105 is essential for T cell antigen receptor-induced proliferation.
Nat Immunol. 2009 Jan;10(1):38-47. doi: 10.1038/ni.1685. Epub 2008 Dec 7.
4
New insights into NF-kappaB regulation and function.
Trends Immunol. 2008 Oct;29(10):469-78. doi: 10.1016/j.it.2008.07.003. Epub 2008 Sep 3.
5
The Cdc14B-Cdh1-Plk1 axis controls the G2 DNA-damage-response checkpoint.
Cell. 2008 Jul 25;134(2):256-67. doi: 10.1016/j.cell.2008.05.043.
6
UV as an amplifier rather than inducer of NF-kappaB activity.
Mol Cell. 2008 Jun 6;30(5):632-41. doi: 10.1016/j.molcel.2008.03.017.
7
Introducing spatial information into predictive NF-kappaB modelling--an agent-based approach.
PLoS One. 2008 Jun 4;3(6):e2367. doi: 10.1371/journal.pone.0002367.
8
Understanding NF-kappaB signaling via mathematical modeling.
Mol Syst Biol. 2008;4:192. doi: 10.1038/msb.2008.30. Epub 2008 May 6.
9
Macromolecular modeling with rosetta.
Annu Rev Biochem. 2008;77:363-82. doi: 10.1146/annurev.biochem.77.062906.171838.
10
Promiscuous mutations activate the noncanonical NF-kappaB pathway in multiple myeloma.
Cancer Cell. 2007 Aug;12(2):131-44. doi: 10.1016/j.ccr.2007.07.003.
Zotero 插件