Claus-Walker J, Carter R E, Di Ferrante M, Singh J
Paraplegia. 1977 Nov;15(3):202-8. doi: 10.1038/sc.1977.30.
Onset of paralysis by cervical spinal cord injury led immediately to temporary adrenocortical activation and, within 2 days, to sustained skin and bone breakdown. Urine cAMP was increased, blood parathyroid hormone, renin activity, and electrolytes were normal, and fluid and electrolytes balance became negative during the initial 6 days of paralysis.
颈脊髓损伤导致的瘫痪发作立即引起肾上腺皮质暂时激活,并在2天内导致皮肤和骨骼持续破坏。尿环磷酸腺苷(cAMP)增加,血甲状旁腺激素、肾素活性和电解质正常,在瘫痪的最初6天内液体和电解质平衡呈负平衡。