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本文引用的文献

1
MEPE's diverse effects on mineralization.MEPE 对矿化的多种影响。
Calcif Tissue Int. 2010 Jan;86(1):42-6. doi: 10.1007/s00223-009-9313-z. Epub 2009 Dec 9.
2
Twenty bone-mineral-density loci identified by large-scale meta-analysis of genome-wide association studies.通过全基因组关联研究的大规模荟萃分析确定的20个骨密度位点。
Nat Genet. 2009 Nov;41(11):1199-206. doi: 10.1038/ng.446. Epub 2009 Oct 4.
3
Phosphorylation-dependent inhibition of mineralization by osteopontin ASARM peptides is regulated by PHEX cleavage.骨桥蛋白 ASARM 肽通过磷酸化依赖性抑制矿化,受 PHEX 切割调节。
J Bone Miner Res. 2010 Apr;25(4):695-705. doi: 10.1359/jbmr.090832.
4
Matrix extracellular phosphoglycoprotein (MEPE) is a new bone renal hormone and vascularization modulator.基质细胞外磷酸糖蛋白(MEPE)是一种新型骨肾激素和血管生成调节剂。
Endocrinology. 2009 Sep;150(9):4012-23. doi: 10.1210/en.2009-0216. Epub 2009 Jun 11.
5
Endocrine functions of bone in mineral metabolism regulation.骨骼在矿物质代谢调节中的内分泌功能。
J Clin Invest. 2008 Dec;118(12):3820-8. doi: 10.1172/JCI36479. Epub 2008 Dec 1.
6
MEPE-ASARM peptides control extracellular matrix mineralization by binding to hydroxyapatite: an inhibition regulated by PHEX cleavage of ASARM.MEPE-ASARM肽通过与羟基磷灰石结合来控制细胞外基质矿化:一种由ASARM的PHEX裂解调节的抑制作用。
J Bone Miner Res. 2008 Oct;23(10):1638-49. doi: 10.1359/jbmr.080601.
7
Aberrant Phex function in osteoblasts and osteocytes alone underlies murine X-linked hypophosphatemia.仅成骨细胞和骨细胞中异常的Phex功能是小鼠X连锁低磷血症的基础。
J Clin Invest. 2008 Feb;118(2):722-34. doi: 10.1172/JCI32702.
8
Degradation of MEPE, DMP1, and release of SIBLING ASARM-peptides (minhibins): ASARM-peptide(s) are directly responsible for defective mineralization in HYP.MEPE、DMP1的降解以及SIBLING ASARM肽(微小抑制素)的释放:ASARM肽直接导致低磷性骨软化症中的矿化缺陷。
Endocrinology. 2008 Apr;149(4):1757-72. doi: 10.1210/en.2007-1205. Epub 2007 Dec 27.
9
Phosphorylated acidic serine-aspartate-rich MEPE-associated motif peptide from matrix extracellular phosphoglycoprotein inhibits phosphate regulating gene with homologies to endopeptidases on the X-chromosome enzyme activity.来自基质细胞外磷酸糖蛋白的磷酸化富含酸性丝氨酸-天冬氨酸的MEPE相关基序肽抑制与X染色体上内肽酶具有同源性的磷酸盐调节基因的酶活性。
J Endocrinol. 2007 Jan;192(1):261-7. doi: 10.1677/joe.1.07059.
10
Klotho converts canonical FGF receptor into a specific receptor for FGF23.α-klotho将经典成纤维细胞生长因子受体转化为成纤维细胞生长因子23的特异性受体。
Nature. 2006 Dec 7;444(7120):770-4. doi: 10.1038/nature05315. Epub 2006 Oct 29.

ASARM mineralization hypothesis: a bridge too far?

作者信息

David Valentin, Quarles L Darryl

机构信息

Department of Medicine, Division of Nephrology, University of Tennessee Health Science Center, Memphis, TN 38163, USA.

出版信息

J Bone Miner Res. 2010 Apr;25(4):692-4. doi: 10.1002/jbmr.69.

DOI:10.1002/jbmr.69
PMID:20200985
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3153325/
Abstract
摘要