相似文献
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To build a synapse: signaling pathways in neuromuscular junction assembly.建立突触：神经肌肉接头组装中的信号通路。
Development. 2010 Apr;137(7):1017-33. doi: 10.1242/dev.038711.
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Glia cell line-derived neurotrophic factor regulates the distribution of acetylcholine receptors in mouse primary skeletal muscle cells.胶质细胞系源性神经营养因子调节小鼠原代骨骼肌细胞中乙酰胆碱受体的分布。
Neuroscience. 2004;128(3):497-509. doi: 10.1016/j.neuroscience.2004.06.067.
3
The neuromuscular junction: selective remodeling of synaptic regulators at the nerve/muscle interface.神经肌肉接头：神经/肌肉界面处突触调节蛋白的选择性重塑。
Mech Dev. 2013 Jun-Aug;130(6-8):402-11. doi: 10.1016/j.mod.2012.09.004. Epub 2012 Sep 29.
4
New dogs in the dogma: Lrp4 and Tid1 in neuromuscular synapse formation.教条中的新成员：Lrp4和Tid1在神经肌肉突触形成中的作用
Neuron. 2008 Nov 26;60(4):526-8. doi: 10.1016/j.neuron.2008.11.003.
5
Molecular dissection of neuromuscular junction formation.神经肌肉接头形成的分子解析
Trends Neurosci. 2003 Jul;26(7):335-7. doi: 10.1016/S0166-2236(03)00131-0.
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LRP4 serves as a coreceptor of agrin.低密度脂蛋白受体相关蛋白4（LRP4）作为聚集蛋白聚糖的共受体。
Neuron. 2008 Oct 23;60(2):285-97. doi: 10.1016/j.neuron.2008.10.006.
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Neural agrin increases postsynaptic ACh receptor packing by elevating rapsyn protein at the mouse neuromuscular synapse.神经聚集蛋白通过提高小鼠神经肌肉突触处的rapsyn蛋白水平，增加突触后乙酰胆碱受体的聚集。
Dev Neurobiol. 2008 Aug;68(9):1153-69. doi: 10.1002/dneu.20654.
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A novel pathway for MuSK to induce key genes in neuromuscular synapse formation.一种MuSK诱导神经肌肉突触形成关键基因的新途径。
J Cell Biol. 2003 May 26;161(4):727-36. doi: 10.1083/jcb.200210156. Epub 2003 May 19.
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The muscle protein Dok-7 is essential for neuromuscular synaptogenesis.肌肉蛋白Dok-7对神经肌肉突触形成至关重要。
Science. 2006 Jun 23;312(5781):1802-5. doi: 10.1126/science.1127142.
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Tyrosine phosphatases such as SHP-2 act in a balance with Src-family kinases in stabilization of postsynaptic clusters of acetylcholine receptors.酪氨酸磷酸酶（如SHP-2）在与Src家族激酶的平衡中发挥作用，以稳定乙酰胆碱受体的突触后簇。
BMC Neurosci. 2007 Jul 2;8:46. doi: 10.1186/1471-2202-8-46.
引用本文的文献
1
Synergic action of MicroRNAs and Wnts delivered by motor neuron EVs in promoting AChR clustering.运动神经元细胞外囊泡传递的微小RNA和Wnt信号在促进乙酰胆碱受体聚集方面的协同作用。
Cell Commun Signal. 2025 Aug 1;23(1):360. doi: 10.1186/s12964-025-02312-x.
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Tissue-engineered neuromuscular organoids.组织工程化神经肌肉类器官
Commun Biol. 2025 Jul 19;8(1):1074. doi: 10.1038/s42003-025-08484-z.
3
Molecular organization of central cholinergic synapses.中枢胆碱能突触的分子组织
Proc Natl Acad Sci U S A. 2025 Apr 29;122(17):e2422173122. doi: 10.1073/pnas.2422173122. Epub 2025 Apr 24.
4
Regulation of miR-206 in denervated and dystrophic muscles, and its effect on acetylcholine receptor clustering.miR-206在失神经和营养不良肌肉中的调控及其对乙酰胆碱受体聚集的影响。
J Cell Sci. 2024 Dec 15;137(24). doi: 10.1242/jcs.262303. Epub 2024 Dec 13.
5
Therapeutic applications and challenges in myostatin inhibition for enhanced skeletal muscle mass and functions.抑制肌肉生长抑制素以增加骨骼肌质量和功能的治疗应用及挑战。
Mol Cell Biochem. 2025 Mar;480(3):1535-1553. doi: 10.1007/s11010-024-05120-y. Epub 2024 Sep 28.
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BDNF/TrkB signalling, in cooperation with muscarinic signalling, retrogradely regulates PKA pathway to phosphorylate SNAP-25 and Synapsin-1 at the neuromuscular junction.脑源性神经营养因子/TrkB 信号与毒蕈碱信号协同作用，逆行调节蛋白激酶 A 通路，使突触相关蛋白 25 和突触素 1 在神经肌肉接头处发生磷酸化。
Cell Commun Signal. 2024 Jul 23;22(1):371. doi: 10.1186/s12964-024-01735-2.
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Aberrant evoked calcium signaling and nAChR cluster morphology in a D90A hiPSC-derived neuromuscular model.D90A人诱导多能干细胞衍生的神经肌肉模型中异常的诱发钙信号和烟碱型乙酰胆碱受体簇形态
Front Cell Dev Biol. 2024 Jun 20;12:1429759. doi: 10.3389/fcell.2024.1429759. eCollection 2024.
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The MuSK agonist antibody protects the neuromuscular junction and extends the lifespan in C9orf72-ALS mice.MuSK 激动剂抗体可保护神经肌肉接头并延长 C9orf72-ALS 小鼠的寿命。
Mol Ther. 2024 Jul 3;32(7):2176-2189. doi: 10.1016/j.ymthe.2024.05.016. Epub 2024 May 11.
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Impaired communication at the neuromotor axis during Degenerative Cervical Myelopathy.退行性颈椎脊髓病期间神经运动轴的通讯受损。
Front Cell Neurosci. 2024 Jan 10;17:1316432. doi: 10.3389/fncel.2023.1316432. eCollection 2023.
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Secreted Metabolome of ALS-Related hSOD1(G93A) Primary Cultures of Myocytes and Implications for Myogenesis.肌细胞 ALS 相关 hSOD1(G93A)原代培养的分泌代谢组学及其对肌发生的影响。
Cells. 2023 Nov 30;12(23):2751. doi: 10.3390/cells12232751.
本文引用的文献
1
Ephexin1 is required for structural maturation and neurotransmission at the neuromuscular junction.Ephexin1 对于神经肌肉接头的结构成熟和神经递质传递是必需的。
Neuron. 2010 Jan 28;65(2):204-16. doi: 10.1016/j.neuron.2010.01.012.
2
Ubiquitin carboxyl-terminal hydrolase L1 is required for maintaining the structure and function of the neuromuscular junction.泛素羧基末端水解酶 L1 对于维持神经肌肉接头的结构和功能是必需的。
Proc Natl Acad Sci U S A. 2010 Jan 26;107(4):1636-41. doi: 10.1073/pnas.0911516107. Epub 2010 Jan 4.
3
A secreted complement-control-related protein ensures acetylcholine receptor clustering.一种分泌型补体控制相关蛋白可确保乙酰胆碱受体聚集。
Nature. 2009 Oct 15;461(7266):992-6. doi: 10.1038/nature08430. Epub 2009 Sep 30.
4
The proteasome-associated deubiquitinating enzyme Usp14 is essential for the maintenance of synaptic ubiquitin levels and the development of neuromuscular junctions.蛋白酶体相关去泛素化酶Usp14对于维持突触泛素水平和神经肌肉接头的发育至关重要。
J Neurosci. 2009 Sep 2;29(35):10909-19. doi: 10.1523/JNEUROSCI.2635-09.2009.
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Glia and muscle sculpt neuromuscular arbors by engulfing destabilized synaptic boutons and shed presynaptic debris.神经胶质细胞和肌肉通过吞噬不稳定的突触小体和清除突触前碎片来塑造神经肌肉树突。
PLoS Biol. 2009 Aug;7(8):e1000184. doi: 10.1371/journal.pbio.1000184. Epub 2009 Aug 25.
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Regulation of acetylcholine receptor clustering by ADF/cofilin-directed vesicular trafficking.由ADF/丝切蛋白介导的囊泡运输对乙酰胆碱受体聚集的调控
Nat Neurosci. 2009 Jul;12(7):848-56. doi: 10.1038/nn.2322. Epub 2009 May 31.
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The translational repressors Nanos and Pumilio have divergent effects on presynaptic terminal growth and postsynaptic glutamate receptor subunit composition.翻译抑制因子Nanos和Pumilio对突触前终末生长和突触后谷氨酸受体亚基组成具有不同影响。
J Neurosci. 2009 Apr 29;29(17):5558-72. doi: 10.1523/JNEUROSCI.0520-09.2009.
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Fbxo45 forms a novel ubiquitin ligase complex and is required for neuronal development.Fbxo45形成一种新型泛素连接酶复合物，是神经元发育所必需的。
Mol Cell Biol. 2009 Jul;29(13):3529-43. doi: 10.1128/MCB.00364-09. Epub 2009 Apr 27.
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Aberrant development of neuromuscular junctions in glycosylation-defective Large(myd) mice.糖基化缺陷型Large（myd）小鼠神经肌肉接头的异常发育
Neuromuscul Disord. 2009 May;19(5):366-78. doi: 10.1016/j.nmd.2009.02.011. Epub 2009 Apr 5.
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Abnormal development of the neuromuscular junction in Nedd4-deficient mice.Nedd4基因缺陷小鼠神经肌肉接头的异常发育。
Dev Biol. 2009 Jun 1;330(1):153-66. doi: 10.1016/j.ydbio.2009.03.023. Epub 2009 Apr 1.
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