Lambert C R
Division of Cardiology, University of South Alabama Medical Center, Mobile.
Cardiol Clin. 1991 Feb;9(1):1-10.
The pathophysiology of stable angina is discussed with respect to anatomic substrate, coronary and systemic hemodynamic mechanisms, the dynamic nature of coronary artery stenoses, and determinants of myocardial oxygen consumption. Cellular mechanisms involved in ischemia and the transduction of these changes into angina are also reviewed.
本文从解剖学基础、冠状动脉和全身血流动力学机制、冠状动脉狭窄的动态特性以及心肌氧消耗的决定因素等方面探讨了稳定型心绞痛的病理生理学。文中还综述了缺血相关的细胞机制以及这些变化转化为心绞痛的传导过程。
