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Guillain-Barré syndrome after short-course efalizumab treatment.

作者信息

Turatti Marco, Tamburin Stefano, Idone Domenico, Praitano Maria Luigia, Zanette Giampietro

出版信息

J Neurol. 2010 Aug;257(8):1404-5. doi: 10.1007/s00415-010-5521-8. Epub 2010 Mar 27.

DOI:10.1007/s00415-010-5521-8
PMID:20349079
Abstract
摘要

相似文献

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Guillain-Barré syndrome after short-course efalizumab treatment.
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Carbohydrate mimicry between human ganglioside GM1 and Campylobacter jejuni lipooligosaccharide causes Guillain-Barre syndrome.人神经节苷脂GM1与空肠弯曲菌脂寡糖之间的碳水化合物模拟导致格林-巴利综合征。
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Guillain-Barré syndrome after use of alemtuzumab (Campath) in a patient with T-cell prolymphocytic leukemia: a case report and review of the literature.使用阿仑单抗(Campath)治疗T细胞幼淋巴细胞白血病患者后发生吉兰-巴雷综合征:一例病例报告及文献复习
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Pharmacological treatment other than corticosteroids, intravenous immunoglobulin and plasma exchange for Guillain-Barré syndrome.除皮质类固醇、静脉注射免疫球蛋白和血浆置换外,用于吉兰-巴雷综合征的药物治疗。
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Severe meningo-radiculo-neuritis associated with ipilimumab.与伊匹单抗相关的严重脑膜-脊神经根神经炎。
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本文引用的文献

1
A case of natural killer cell monoclonal expansion during efalizumab treatment in a patient with psoriasis.1例银屑病患者使用依法利珠单抗治疗期间自然杀伤细胞单克隆扩增的病例。
Br J Dermatol. 2009 Apr;160(4):896-7. doi: 10.1111/j.1365-2133.2009.09059.x. Epub 2009 Feb 23.
2
Efalizumab-associated Guillain-Barre syndrome.
Arch Dermatol. 2008 Oct;144(10):1396-7. doi: 10.1001/archderm.144.10.1396.
3
Clinical features, pathogenesis, and treatment of Guillain-Barré syndrome.格林-巴利综合征的临床特征、发病机制及治疗
Lancet Neurol. 2008 Oct;7(10):939-50. doi: 10.1016/S1474-4422(08)70215-1.
4
Tumor necrosis factor-alpha antagonists and neuropathy.肿瘤坏死因子-α拮抗剂与神经病变
Muscle Nerve. 2008 Mar;37(3):281-92. doi: 10.1002/mus.20924.
5
Non-T(H)1 cytokines are augmented systematically early in Guillain-Barré syndrome.在吉兰-巴雷综合征早期,非辅助性T细胞1(Th1)细胞因子会系统性增加。
Neurology. 2002 Feb 12;58(3):476-8. doi: 10.1212/wnl.58.3.476.
6
Approaches for identifying and defining environmentally associated rheumatic disorders.
Arthritis Rheum. 2000 Feb;43(2):243-9. doi: 10.1002/1529-0131(200002)43:2<243::AID-ANR2>3.0.CO;2-K.