相似文献
1
The RING finger domain of Varicella-Zoster virus ORF61p has E3 ubiquitin ligase activity that is essential for efficient autoubiquitination and dispersion of Sp100-containing nuclear bodies.
J Virol. 2010 Jul;84(13):6861-5. doi: 10.1128/JVI.00335-10. Epub 2010 Apr 14.
2
Complementation of a herpes simplex virus ICP0 null mutant by varicella-zoster virus ORF61p.
J Virol. 2009 Oct;83(20):10637-43. doi: 10.1128/JVI.01144-09. Epub 2009 Aug 5.
3
Components of nuclear domain 10 bodies regulate varicella-zoster virus replication.
J Virol. 2009 May;83(9):4262-74. doi: 10.1128/JVI.00021-09. Epub 2009 Feb 11.
4
Comparison of the biological and biochemical activities of several members of the alphaherpesvirus ICP0 family of proteins.
J Virol. 2010 Apr;84(7):3476-87. doi: 10.1128/JVI.02544-09. Epub 2010 Jan 27.
5
Alphaherpesvirus proteins related to herpes simplex virus type 1 ICP0 affect cellular structures and proteins.
J Virol. 2000 Nov;74(21):10006-17. doi: 10.1128/jvi.74.21.10006-10017.2000.
6
The cellular localization pattern of Varicella-Zoster virus ORF29p is influenced by proteasome-mediated degradation.
J Virol. 2006 Feb;80(3):1497-512. doi: 10.1128/JVI.80.3.1497-1512.2006.
7
Identification of three redundant segments responsible for herpes simplex virus 1 ICP0 to fuse with ND10 nuclear bodies.
J Virol. 2015 Apr;89(8):4214-26. doi: 10.1128/JVI.03658-14. Epub 2015 Jan 28.
8
PML plays both inimical and beneficial roles in HSV-1 replication.
Proc Natl Acad Sci U S A. 2016 May 24;113(21):E3022-8. doi: 10.1073/pnas.1605513113. Epub 2016 May 9.
9
N-terminal phosphorylation sites of herpes simplex virus 1 ICP0 differentially regulate its activities and enhance viral replication.
J Virol. 2013 Feb;87(4):2109-19. doi: 10.1128/JVI.02588-12. Epub 2012 Dec 5.
10
The degradation of promyelocytic leukemia and Sp100 proteins by herpes simplex virus 1 is mediated by the ubiquitin-conjugating enzyme UbcH5a.
Proc Natl Acad Sci U S A. 2003 Jul 22;100(15):8963-8. doi: 10.1073/pnas.1533420100. Epub 2003 Jul 10.
引用本文的文献
1
PML Body Component Sp100A Restricts Wild-Type Herpes Simplex Virus 1 Infection.
J Virol. 2022 Apr 27;96(8):e0027922. doi: 10.1128/jvi.00279-22. Epub 2022 Mar 30.
2
Varicella Zoster Virus Neuronal Latency and Reactivation Modeled in Vitro.
Curr Top Microbiol Immunol. 2023;438:103-134. doi: 10.1007/82_2021_244.
3
A Tale of Usurpation and Subversion: SUMO-Dependent Integrity of Promyelocytic Leukemia Nuclear Bodies at the Crossroad of Infection and Immunity.
Front Cell Dev Biol. 2021 Aug 27;9:696234. doi: 10.3389/fcell.2021.696234. eCollection 2021.
4
TTC3-Mediated Protein Quality Control, A Potential Mechanism for Cognitive Impairment.
Cell Mol Neurobiol. 2022 Aug;42(6):1659-1669. doi: 10.1007/s10571-021-01060-z. Epub 2021 Feb 27.
5
The Fate of Speckled Protein 100 (Sp100) During Herpesviruses Infection.
Front Cell Infect Microbiol. 2021 Feb 1;10:607526. doi: 10.3389/fcimb.2020.607526. eCollection 2020.
6
The Role of ND10 Nuclear Bodies in Herpesvirus Infection: A Frenemy for the Virus?
Viruses. 2021 Feb 3;13(2):239. doi: 10.3390/v13020239.
7
The Speckled Protein (SP) Family: Immunity's Chromatin Readers.
Trends Immunol. 2020 Jul;41(7):572-585. doi: 10.1016/j.it.2020.04.007. Epub 2020 May 5.
8
Pseudorabies Virus UL24 Abrogates Tumor Necrosis Factor Alpha-Induced NF-κB Activation by Degrading P65.
Viruses. 2020 Jan 2;12(1):51. doi: 10.3390/v12010051.
9
Discovery of Small-Molecule Inhibitors Targeting the E3 Ubiquitin Ligase Activity of the Herpes Simplex Virus 1 ICP0 Protein Using an High-Throughput Screening Assay.
J Virol. 2019 Jun 14;93(13). doi: 10.1128/JVI.00619-19. Print 2019 Jul 1.
10
Sp100 colocalizes with HPV replication foci and restricts the productive stage of the infectious cycle.
PLoS Pathog. 2017 Oct 2;13(10):e1006660. doi: 10.1371/journal.ppat.1006660. eCollection 2017 Oct.
本文引用的文献
1
Comparison of the biological and biochemical activities of several members of the alphaherpesvirus ICP0 family of proteins.
J Virol. 2010 Apr;84(7):3476-87. doi: 10.1128/JVI.02544-09. Epub 2010 Jan 27.
2
Histone deacetylases 1 and 2 are phosphorylated at novel sites during varicella-zoster virus infection.
J Virol. 2009 Nov;83(22):11502-13. doi: 10.1128/JVI.01318-09. Epub 2009 Sep 9.
3
Complementation of a herpes simplex virus ICP0 null mutant by varicella-zoster virus ORF61p.
J Virol. 2009 Oct;83(20):10637-43. doi: 10.1128/JVI.01144-09. Epub 2009 Aug 5.
4
Regulation of the ORF61 promoter and ORF61 functions in varicella-zoster virus replication and pathogenesis.
J Virol. 2009 Aug;83(15):7560-72. doi: 10.1128/JVI.00118-09. Epub 2009 May 20.
5
Components of nuclear domain 10 bodies regulate varicella-zoster virus replication.
J Virol. 2009 May;83(9):4262-74. doi: 10.1128/JVI.00021-09. Epub 2009 Feb 11.
6
The co-chaperone BAG3 regulates Herpes Simplex Virus replication.
Proc Natl Acad Sci U S A. 2008 Dec 30;105(52):20912-7. doi: 10.1073/pnas.0810656105. Epub 2008 Dec 16.
7
Nuclear import of the varicella-zoster virus latency-associated protein ORF63 in primary neurons requires expression of the lytic protein ORF61 and occurs in a proteasome-dependent manner.
J Virol. 2008 Sep;82(17):8673-86. doi: 10.1128/JVI.00685-08. Epub 2008 Jun 18.
8
Posttranslational modification and cell type-specific degradation of varicella-zoster virus ORF29p.
J Virol. 2006 Nov;80(21):10836-46. doi: 10.1128/JVI.00966-06. Epub 2006 Sep 6.
9
Evidence for a role of the cellular ND10 protein PML in mediating intrinsic immunity against human cytomegalovirus infections.
J Virol. 2006 Aug;80(16):8006-18. doi: 10.1128/JVI.00743-06.
10
PML contributes to a cellular mechanism of repression of herpes simplex virus type 1 infection that is inactivated by ICP0.
J Virol. 2006 Aug;80(16):7995-8005. doi: 10.1128/JVI.00734-06.
Zotero 插件