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1
Roles of trinucleotide-repeat RNA in neurological disease and degeneration.
Trends Neurosci. 2010 Jun;33(6):292-8. doi: 10.1016/j.tins.2010.03.004. Epub 2010 Apr 14.
2
The molecular pathogenesis of repeat expansion diseases.
Biochem Soc Trans. 2022 Feb 28;50(1):119-134. doi: 10.1042/BST20200143.
3
RNA toxicity and foci formation in microsatellite expansion diseases.
Curr Opin Genet Dev. 2017 Jun;44:17-29. doi: 10.1016/j.gde.2017.01.005. Epub 2017 Feb 14.
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Trinucleotide repeat expansion in neurological disease.
Ann Neurol. 1994 Dec;36(6):814-22. doi: 10.1002/ana.410360604.
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CAG-polyglutamine-repeat mutations: independence from gene context.
Philos Trans R Soc Lond B Biol Sci. 1999 Jun 29;354(1386):1083-8. doi: 10.1098/rstb.1999.0463.
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The unstable repeats--three evolving faces of neurological disease.
Neuron. 2013 Mar 6;77(5):825-43. doi: 10.1016/j.neuron.2013.02.022.
7
Exploring the Potential of Small Molecule-Based Therapeutic Approaches for Targeting Trinucleotide Repeat Disorders.
Mol Neurobiol. 2020 Jan;57(1):566-584. doi: 10.1007/s12035-019-01724-4. Epub 2019 Aug 9.
8
Recent advances in molecular therapies for neurological disease: triplet repeat disorders.
Hum Mol Genet. 2019 Oct 1;28(R1):R80-R87. doi: 10.1093/hmg/ddz138.
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Cellular toxicity of expanded RNA repeats: focus on RNA foci.
Hum Mol Genet. 2011 Oct 1;20(19):3811-21. doi: 10.1093/hmg/ddr299. Epub 2011 Jul 4.
10
[Research progress in roles of microRNA in polyglutamine diseases].
Zhonghua Yi Xue Yi Chuan Xue Za Zhi. 2010 Aug;27(4):406-9. doi: 10.3760/cma.j.issn.1003-9406.2010.04.010.

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2
Adaptation of RiPCA for the Live-Cell Detection of mRNA-Protein Interactions.
Biochemistry. 2023 Dec 5;62(23):3323-3336. doi: 10.1021/acs.biochem.3c00334. Epub 2023 Nov 14.
3
Two Decades of Arrayed Imaging Reflectometry for Sensitive, High-Throughput Biosensing.
Biosensors (Basel). 2023 Sep 5;13(9):870. doi: 10.3390/bios13090870.
5
Biomolecular condensates: Formation mechanisms, biological functions, and therapeutic targets.
MedComm (2020). 2023 Feb 28;4(2):e223. doi: 10.1002/mco2.223. eCollection 2023 Apr.
6
Compendious survey of protein tandem repeats in inbred mouse strains.
BMC Genom Data. 2022 Aug 5;23(1):62. doi: 10.1186/s12863-022-01079-1.
8
as a Model of Unconventional Translation in Spinocerebellar Ataxia Type 3.
Cells. 2022 Apr 4;11(7):1223. doi: 10.3390/cells11071223.
9
Molecular conformations and dynamics of nucleotide repeats associated with neurodegenerative diseases: double helices and CAG hairpin loops.
Comput Struct Biotechnol J. 2021 Apr 26;19:2819-2832. doi: 10.1016/j.csbj.2021.04.037. eCollection 2021.
10
Advances in Pluripotent Stem Cells: History, Mechanisms, Technologies, and Applications.
Stem Cell Rev Rep. 2020 Feb;16(1):3-32. doi: 10.1007/s12015-019-09935-x.

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Pentamidine reverses the splicing defects associated with myotonic dystrophy.
Proc Natl Acad Sci U S A. 2009 Nov 3;106(44):18551-6. doi: 10.1073/pnas.0903234106. Epub 2009 Oct 12.
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RNA gain-of-function in spinocerebellar ataxia type 8.
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Triplet-repeat oligonucleotide-mediated reversal of RNA toxicity in myotonic dystrophy.
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Reversal of RNA dominance by displacement of protein sequestered on triplet repeat RNA.
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Fragile X syndrome: loss of local mRNA regulation alters synaptic development and function.
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Ubiquitin, the proteasome and protein degradation in neuronal function and dysfunction.
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miR-19, miR-101 and miR-130 co-regulate ATXN1 levels to potentially modulate SCA1 pathogenesis.
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RNA toxicity is a component of ataxin-3 degeneration in Drosophila.
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MBNL1 associates with YB-1 in cytoplasmic stress granules.
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