Seliger G M, Lefever F, Lukas R, Chen J, Schwartz S, Codeghini L, Abrams G
Department of Neurology, Helen Hayes Hospital, West Haverstraw 10993.
Brain Inj. 1991 Jan-Mar;5(1):53-6. doi: 10.3109/02699059108998511.
Pure word deafness, usually involving left hemisphere focal lesions that destroy Heschl's gyrus and/or isolate auditory association cortex, may be rare, but cases with additional perceptual or cognitive symptoms may not be. Word deafness can be transient or evolving, and has been seen in various conditions without identifiable focal lesions. Only two closed head injury cases with focal contusions have been reported; we report two more, with diffuse damage and no focal signs. One patient's symptoms evolved soon after they were recognized. The other patient's unresponsiveness to spoken verbal stimuli persisted despite relatively preserved reading and speech, in a context of poor initiation and moderately severe cognitive impairment. Unrecognized, word deafness following head injury could lead to overestimation of coma duration if transient, and impede rehabilitation if chronic.
纯词聋通常涉及左半球局灶性病变，这些病变会破坏颞横回和/或隔离听觉联合皮层，可能较为罕见，但伴有其他感知或认知症状的病例可能并非如此。词聋可以是短暂的或渐进性的，并且在没有可识别局灶性病变的各种情况下都有出现。仅报告了两例伴有局灶性挫伤的闭合性颅脑损伤病例；我们又报告了另外两例，伴有弥漫性损伤且无局灶性体征。一名患者的症状在被发现后不久就有所发展。另一名患者尽管阅读和言语相对保留，但在起始能力差和中度严重认知障碍的情况下，对言语刺激仍无反应。如果未被识别，头部受伤后的词聋如果是短暂的，可能会导致对昏迷持续时间的高估，如果是慢性的，则会阻碍康复。
