Sokrut V N, Iabluchanskiĭ N I, Shvirenko I R
Fiziol Zh (1978). 1991 Mar-Apr;37(2):31-6.
Experiment on 72 dogs has shown that myocardial infarction (M1) against a background of hyperglycemia proceeds as shifts healing hyperactive MI. Shifts in carbohydrate metabolism results in disturbances of dynamics of the necrotic infarction zone processes that induces complications of healing changes in the content of myoglobin, creatine kinase, aspartateaminotransferase with MI against a background of hyperglycemia greatly differ from those typical of noncomplicated and complicated hyperactive MI.
对72只狗进行的实验表明,在高血糖背景下发生的心肌梗死(MI)呈现为愈合亢进型心肌梗死的变化。碳水化合物代谢的改变导致坏死梗死区进程动态的紊乱,进而引发愈合变化的并发症。在高血糖背景下,心肌梗死时肌红蛋白、肌酸激酶、天冬氨酸转氨酶含量的变化与非复杂性和复杂性愈合亢进型心肌梗死的典型变化有很大不同。