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声带注射诱导的三维杓状软骨运动

Three-dimensional arytenoid movement induced by vocal fold injections.

机构信息

Clinical Center for Voice Care, Department of Otolaryngology-Head and Neck Surgery, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

出版信息

Laryngoscope. 2010 Aug;120(8):1563-8. doi: 10.1002/lary.20973.

DOI:10.1002/lary.20973
PMID:20564669
Abstract

OBJECTIVES/HYPOTHESIS: To quantitatively characterize arytenoid movement induced by vocal fold injection augmentation in an excised larynx model.

STUDY DESIGN

: Laboratory and computational.

METHODS

Vocal folds of human cadaveric larynges were injected with calcium hydroxylapatite. High-resolution computed tomography scans were obtained before and after injection. Densities corresponding to the arytenoid and cricoid cartilages were extracted and processed with custom MATLAB routines to generate selective three-dimensional reconstructions of the larynx. Pre- and postinjection positions of the arytenoid were compared.

RESULTS

Vocal fold injections resulted in predominantly small-magnitude medial rotation and medial translation of the arytenoid. Movements in other directions as would be expected in physiologic adduction were not observed.

CONCLUSIONS

Vocal fold injection augmentation induced passive movement of the arytenoid that has not been described previously. This movement does not reproduce the trajectory of physiologic adduction. This finding has implications for the treatment of unilateral vocal fold paralysis without arytenoid repositioning maneuvers.

摘要

目的/假设:定量描述在离体喉模型中声带注射增强引起的杓状软骨运动。

研究设计

实验室和计算。

方法

用人尸体喉的声带进行羟基磷灰石注射。在注射前后获得高分辨率计算机断层扫描。提取对应杓状软骨和环状软骨的密度,并使用自定义 MATLAB 例程进行处理,生成喉的选择性三维重建。比较注射前后杓状软骨的位置。

结果

声带注射主要导致杓状软骨的小幅度内旋和内移。未观察到预期的生理内收的其他方向的运动。

结论

声带注射增强引起了杓状软骨的被动运动,这是以前没有描述过的。这种运动没有复制生理内收的轨迹。这一发现对不进行杓状软骨复位术治疗单侧声带麻痹具有重要意义。

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Three-dimensional conformation of the injected bolus in vocal fold injections in a cadaver model.
尸体模型中声带注射时注射物团块的三维形态。
Otolaryngol Head Neck Surg. 2011 Apr;144(4):552-7. doi: 10.1177/0194599810395107. Epub 2011 Feb 1.