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本文引用的文献

1
Experimental stress in inflammatory rheumatic diseases: a review of psychophysiological stress responses.炎症性风湿性疾病的实验性应激:心理生理应激反应综述。
Arthritis Res Ther. 2010;12(3):R89. doi: 10.1186/ar3016. Epub 2010 May 17.
2
Depression, inflammation, and pain in patients with rheumatoid arthritis.类风湿关节炎患者的抑郁、炎症与疼痛
Arthritis Rheum. 2009 Aug 15;61(8):1018-24. doi: 10.1002/art.24647.
3
Cumulative childhood stress and autoimmune diseases in adults.成年人童年期累积应激与自身免疫性疾病
Psychosom Med. 2009 Feb;71(2):243-50. doi: 10.1097/PSY.0b013e3181907888. Epub 2009 Feb 2.
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Duloxetine and other antidepressants in the treatment of patients with fibromyalgia.度洛西汀及其他抗抑郁药治疗纤维肌痛患者
Pain Med. 2007 Sep;8 Suppl 2:S63-74. doi: 10.1111/j.1526-4637.2006.00178.x.
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Cerebrospinal fluid corticotropin-releasing factor concentration is associated with pain but not fatigue symptoms in patients with fibromyalgia.在纤维肌痛患者中,脑脊液促肾上腺皮质激素释放因子浓度与疼痛相关,但与疲劳症状无关。
Neuropsychopharmacology. 2006 Dec;31(12):2776-82. doi: 10.1038/sj.npp.1301200. Epub 2006 Aug 23.
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Stress in autoimmune disease models.自身免疫性疾病模型中的应激
Ann N Y Acad Sci. 2006 Jun;1069:51-61. doi: 10.1196/annals.1351.005.
7
The relationship between depression, clinical pain, and experimental pain in a chronic pain cohort.慢性疼痛队列中抑郁症、临床疼痛与实验性疼痛之间的关系。
Arthritis Rheum. 2005 May;52(5):1577-84. doi: 10.1002/art.21008.
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Posttraumatic stress disorder and physical illness: results from clinical and epidemiologic studies.创伤后应激障碍与躯体疾病:临床及流行病学研究结果
Ann N Y Acad Sci. 2004 Dec;1032:141-53. doi: 10.1196/annals.1314.011.
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Influence of social support and emotional context on pain processing and magnetic brain responses in fibromyalgia.社会支持和情绪背景对纤维肌痛患者疼痛处理及脑磁反应的影响
Arthritis Rheum. 2004 Dec;50(12):4035-44. doi: 10.1002/art.20660.
10
Pain catastrophizing and neural responses to pain among persons with fibromyalgia.纤维肌痛患者的疼痛灾难化思维及对疼痛的神经反应
Brain. 2004 Apr;127(Pt 4):835-43. doi: 10.1093/brain/awh098. Epub 2004 Feb 11.

压力在风湿性疾病中的作用。

The role of stress in rheumatic diseases.

出版信息

Arthritis Res Ther. 2010;12(3):123. doi: 10.1186/ar3024. Epub 2010 Jun 7.

DOI:10.1186/ar3024
PMID:20587002
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2911881/
Abstract

Rheumatology patients frequently note the occurrence of stressful or traumatic life events prior to the onset of their illness and/or a relationship between stress and disease flares. For our patients, identifying causal events could represent an effort to give meaning to a chronic and often disabling disease, while noting a link between stress and flares may proffer a sense of control. Whatever purpose the report of stress as an etiological or maintaining factor may serve, the science exploring a causal relationship between stress and autoimmune disease onset and course is expanding. Moreover, stress can also induce symptoms such as pain via nonimmunological mechanisms.

摘要

风湿病患者经常注意到在疾病发作之前发生的紧张或创伤性生活事件,和/或压力与疾病发作之间的关系。对于我们的患者来说,确定因果事件可能代表着对一种慢性且常常使人丧失能力的疾病赋予意义的努力,同时注意到压力与发作之间的联系可能提供一种控制感。无论报告压力作为病因或维持因素的目的是什么,探索压力与自身免疫性疾病发病和病程之间因果关系的科学正在不断发展。此外,压力也可以通过非免疫机制引起疼痛等症状。