Danis Iu, Tsimbolaĭtite Iu, Cherniauskene L, Naktinis I
Farmakol Toksikol. 1978 Mar-Apr;41(2):227-9.
The EPR signal intensity at g 2.0057 was measured in tissues of albino mice subjected to the action of sodium nitrite. An increased signal in the blood and a decreased one in the liver and spleen were registered, this being accounted for by redistribution of ascorbic acid in the organism due to the action of nitrite.
在受到亚硝酸作用的白化小鼠组织中,测量了g值为2.0057时的电子顺磁共振(EPR)信号强度。结果显示,血液中的信号增强,而肝脏和脾脏中的信号减弱,这是由于亚硝酸盐的作用导致机体中抗坏血酸重新分布所致。