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[肌电位对按需起搏器的抑制作用及其临床意义]

[Inhibition of demand pacemakers by myopotentials and its clinical significance].

作者信息

Luo Z M

机构信息

Chang Zheng Hospital, Second Military Medical College, PLA, Shanghai.

出版信息

Zhonghua Xin Xue Guan Bing Za Zhi. 1990 Oct;18(5):276-8, 319.

PMID:2086191
Abstract

Unipolar demand pacemaker myopotential inhibition by 5 different provocative maneuvers was studied in 48 patients with stable pacing rhythm out of 82 patients with unipolar pacemakers from 3 manufacturers implanted over the right pectoralis major to evaluate the incidence of inhibiting phenomenon and its clinical significance. Transient pacing inhibition occurred in 19 (39.6%) of the 48 patients. Among them 3 (30.0%) of 10 patients with Model 28 (EDWARDS), 6 (31.6%) of 19 patients with Model 30 (EDWARDS), 10 (71.4%) of 14 patients with Model 7550 (ELAMEDICAL), and none of the 5 patients with Model P101 (QINMING). The effective maneuvers were the right hand-to-left shoulder press (63.2% positivity), the uprising from a supine position (21.0% positivity) and the hand-to-hand press (15.8% positivity). No pacemaker myopotential inhibition had been induced by Valsalva maneuver and the left hand-to-right shoulder press. 19 patients with positive myopotential inhibition had pauses of pacemaker function lasting from 1.2 to 3.9 seconds, 13 of them with pauses more than 2 seconds. Of all 48 patients, 9 (18.7%) experienced symptoms of cerebral ischemia due to pacemaker myopotential inhibition during test, among whom 8 had ventricular standstill more than 2 seconds. This showed a close relation between symptoms and duration of the pacing inhibition. The causes of the phenomenon and its prevention were briefly discussed.

摘要

在82例植入了3个厂家生产的单极起搏器(经右胸大肌植入)的患者中,选取48例起搏节律稳定的患者,研究5种不同激发动作对单极按需起搏器肌电位的抑制作用,以评估抑制现象的发生率及其临床意义。48例患者中有19例(39.6%)出现短暂起搏抑制。其中,28型(爱德华兹公司)的10例患者中有3例(30.0%),30型(爱德华兹公司)的19例患者中有6例(31.6%),7550型(伊拉医疗公司)的14例患者中有10例(71.4%),而P101型(秦明公司)的5例患者中无一例出现。有效的激发动作是右手按压左肩(阳性率63.2%)、从仰卧位起身(阳性率21.0%)和双手互压(阳性率15.8%)。瓦尔萨尔瓦动作和左手按压右肩未诱发起搏器肌电位抑制。19例肌电位抑制阳性的患者起搏器功能暂停持续1.2至3.9秒,其中13例暂停超过2秒。在所有48例患者中,9例(18.7%)在测试期间因起搏器肌电位抑制出现脑缺血症状,其中8例心室停搏超过2秒。这表明症状与起搏抑制持续时间密切相关。文中简要讨论了该现象的原因及其预防措施。

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