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抗黑色素瘤免疫治疗:IL-12 家族成员的临床和临床前应用。

Antimelanoma immunotherapy: clinical and preclinical applications of IL-12 family members.

机构信息

Division of Dermatology, Department of Internal Related, Kobe University Graduate School of Medicine, 7-5-1 Kusunoki-cho, Chuo-ku, Kobe 650-0017, Japan.

出版信息

Immunotherapy. 2010 Sep;2(5):697-709. doi: 10.2217/imt.10.46.

Abstract

Malignant melanoma has been considered a prototypical 'immunogenic' tumor through clinical observations, such as the spontaneous regression of primary lesions, their higher incidence in immune-suppressed individuals, and the development of vitiligo after immunotherapy. Among many cytokines, IL-12 is one of the best characterized and the most potent anti-tumor cytokines. Although the systemic application of IL-12 resulted in disappointing results owing to its considerable toxicity, IL-12 is not entirely unusable in the clinical setting. IL-12-related cytokines, IL-23 and IL-27, have also been shown to possess anti-tumor activities in preclinical models. Although belonging to the same cytokine family, IL-12, IL-23 and IL-27 were found to have different anti-tumor mechanisms, adjuvant activity for tumor vaccines and adverse effects in a poorly immunogeneic melanoma model. In addition, their novel activities on melanoma have been clarified. We briefly review the key features of these members of the IL-12 cytokine family and discuss their potential relevance to melanoma immunity and antimelanoma immunotherapy.

摘要

恶性黑素瘤一直被认为是一种典型的“免疫原性”肿瘤,这可以从临床观察中得到证实,例如原发性病变的自发消退、免疫抑制个体中更高的发病率以及免疫治疗后白癜风的发展。在许多细胞因子中,IL-12 是最具特征性和最有效的抗肿瘤细胞因子之一。尽管由于其相当大的毒性,全身性应用 IL-12 导致令人失望的结果,但 IL-12 在临床环境中并非完全不可用。IL-12 相关细胞因子,IL-23 和 IL-27,也已在临床前模型中显示出具有抗肿瘤活性。尽管属于同一细胞因子家族,但 IL-12、IL-23 和 IL-27 被发现具有不同的抗肿瘤机制、对肿瘤疫苗的辅助活性以及在免疫原性差的黑素瘤模型中的不良反应。此外,它们在黑素瘤上的新活性也得到了阐明。我们简要回顾了这些 IL-12 细胞因子家族成员的关键特征,并讨论了它们与黑素瘤免疫和抗黑素瘤免疫治疗的潜在相关性。

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