Smoleński R T, Marlewski M
Department of Biochemistry, Academic Medical School of Gdańsk, Poland.
Acta Biochim Pol. 1990;37(1):153-7.
In chronic renal failure AMP-deaminase operates in the erythrocytes at a much higher velocity than in healthy subjects, with a simultaneous shift from the AMP-adenine-inosine-hypoxanthine pathway (55% and 19%, respectively) to the pathway initiated by AMP-deaminase (45% and 81%, respectively).