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诱导性高胰高血糖素血症对Zucker肥胖大鼠的影响。

The effect of induced hyperglucagonaemia on the Zucker fatty rat.

作者信息

Mahmood H A, Wood P J, Marks V

出版信息

Diabetologia. 1978 Jun;14(6):405-12. doi: 10.1007/BF01228135.

Abstract

The effect of chronic treatment with a long-acting glucagon preparation on liver glucagon and insulin receptors, adenylate cyclase and plasma lipids has been examined in Zucker fatty rats (fa/fa) and their lean littermates (Fa/-). Liver insulin and glucagon receptors were examined using radioreceptor assay techniques. Neither fatty nor lean rats showed any change in insulin receptors after glucagon treatment. Glucagon receptors of the fatty rats showed a 33% drop in the number of the glucagon receptors after glucagon treatment, whilst there was no such change in the lean group. Plasma membranes of the treated fatty rats and their controls bound only 50% as much insulin per mg of liver membrane protein as those of the treated lean rats and their controls. Glucagon treatment raised plasma NEFA in lean rats and reduced them in fatty ones. Plasma cholesterol levels were reduced in both groups of animals as were plasma triglycerides, though to a lesser degree in fatty than in lean animals. Glucagon treatment increased basal and stimulated adenylate cyclase activity in the lean rats and even more so in the fatty ones. The data lend no support to the concept that hypertriglyceridaemia in fatty Zucker rats is a consequence of abnormal glucagon responsiveness.

摘要

长效胰高血糖素制剂长期治疗对Zucker肥胖大鼠(fa/fa)及其瘦的同窝大鼠(Fa/-)肝脏胰高血糖素和胰岛素受体、腺苷酸环化酶及血脂的影响已进行了研究。采用放射受体分析技术检测肝脏胰岛素和胰高血糖素受体。胰高血糖素治疗后,肥胖大鼠和瘦大鼠的胰岛素受体均未显示任何变化。肥胖大鼠的胰高血糖素受体在胰高血糖素治疗后数量下降了33%,而瘦大鼠组则无此变化。治疗后的肥胖大鼠及其对照每毫克肝膜蛋白结合的胰岛素量仅为治疗后的瘦大鼠及其对照的50%。胰高血糖素治疗使瘦大鼠血浆非酯化脂肪酸升高,而使肥胖大鼠血浆非酯化脂肪酸降低。两组动物的血浆胆固醇水平均降低,血浆甘油三酯水平也降低,不过肥胖动物降低的程度小于瘦动物。胰高血糖素治疗增加了瘦大鼠基础及刺激状态下的腺苷酸环化酶活性,在肥胖大鼠中增加得更多。这些数据不支持肥胖Zucker大鼠高甘油三酯血症是胰高血糖素反应异常所致这一观点。

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